Paeds SAQs · acute-care-resuscitation-and-toxicology
Hypovolaemic and haemorrhagic shock — formative SAQs
Two MedVellum formative short-answer questions on hypovolaemic and haemorrhagic shock in children: recognising compensated shock before hypotension, the bleeding-versus-non-bleeding fluid fork, balanced crystalloid aliquots for dehydration, the massive transfusion protocol, tranexamic acid dosing and timing, the lethal triad and damage control resuscitation, and early escalation to surgery and retrieval. The marks and timing support transparent self-assessment. They are not an official board format or pass standard.
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Target exams
SAQ 1 — A dehydrated toddler in shock
Question 1 — 10 formative marks; suggested time 15 minutes [2]
A two-year-old presents with three days of vomiting and profuse watery diarrhoea. From the doorway the child is limp and barely responsive, with sunken eyes, a weak cry, marked recession, cool mottled limbs, a heart rate of 165, a capillary refill of 5 seconds, weak central pulses and a blood pressure that is still in the low-normal range. There is no history of injury or bleeding. [2]
- State what you do in the first 60 seconds and why. (2 marks)
- Explain why a low-normal blood pressure does not exclude shock in this child. (2 marks)
- Outline your fluid resuscitation, including the fluid type, aliquot, expected response and reassessment plan. (3 marks)
- Describe how your fluid strategy would change if this child had major haemorrhage instead of dehydration, and why. (3 marks) [2]
Full-credit answer — SAQ 1
Reveal full-credit answer for SAQ 1
1. First 60 seconds
"This child is critically unwell with signs of shock: altered appearance, increased work of breathing and poor circulation to skin. I call the senior paediatric and resuscitation teams now, name a leader, allocate roles, and bring age- and weight-appropriate equipment and monitoring. I begin the hands-on ABCDE primary survey immediately and treat each threat as I find it. Stabilisation comes before diagnosis." The principle is recognise and call for help, then treat life threats as they are found. I also check bedside glucose because the child has poor intake and is unwell. [1] [2]
2. Blood pressure does not exclude shock
Children compensate for volume loss through tachycardia and vasoconstriction, so blood pressure is maintained until roughly 30 per cent of blood volume is lost. This child already shows poor perfusion: a heart rate of 165, weak central pulses, a five-second capillary refill, cool mottled limbs and altered consciousness. That is decompensated shock in evolution despite a low-normal blood pressure. Waiting for hypotension to diagnose shock is a classic and dangerous error; shock is a whole-circulation diagnosis, never a single blood-pressure number. [2]
3. Fluid resuscitation
Because there is no bleeding, this is non-haemorrhagic hypovolaemic shock, so I resuscitate with a balanced crystalloid such as Hartmann or Plasma-Lyte rather than large volumes of 0.9 per cent saline, which worsens hyperchloraemic acidosis. I give an aliquot of 10 to 20 mL per kilogram over five to twenty minutes. Before each aliquot I state the response I expect (warmer skin, shorter capillary refill, stronger pulses, improved interaction, more urine), and after each I reassess and stop for fluid overload or no benefit. The first-hour total is a ceiling, not a target. The Surviving Sepsis Campaign 2026 children's guideline supports up-front boluses with careful reassessment and attention to fluid balance. [4]
4. Change if this were haemorrhage
If the child had major haemorrhage, the first fluid would change from crystalloid to blood products. I would first control the bleeding with direct pressure, a tourniquet or a pelvic binder as appropriate, then activate the paediatric massive transfusion protocol to deliver balanced components (red cells, plasma and platelets in a ratio near one to one to one). I would give tranexamic acid if the injury was within three hours. I would keep crystalloid to the minimum needed to sustain perfusion while blood arrives, because large crystalloid volumes dilute clotting factors and worsen the lethal triad of hypothermia, acidosis and coagulopathy. The first decision in any shock is bleeding or not bleeding, because that decides the first fluid. [5] [9]
You have read the opening of this SAQ. The complete unit — every section and its primary-source references — is part of the Paediatrics Fellowship fellowship atlas.
References7Show ledgerHide ledger
- [1]Topjian AA, Raymond TT, Atkins D, et al. Part 4: Pediatric Basic and Advanced Life Support 2020 American Heart Association Guidelines for Cardiopulmonary Resuscitation and Emergency Cardiovascular Care Pediatrics, 2021.PMID 33087552
- [2]Bjorklund A, Resch J, Slusher T Pediatric Shock Review Pediatrics in review, 2023.PMID 37777656
- [4]Weiss, Scott L Surviving Sepsis Campaign International Guidelines for the Management of Sepsis and Septic Shock in Children 2026 Pediatric critical care medicine : a journal of the Society of Critical Care Medicine and the World Federation of Pediatric Intensive and Critical Care Societies, 2026.PMID 41869844
- [5]Russell, Russell T Pediatric traumatic hemorrhagic shock consensus conference recommendations The journal of trauma and acute care surgery, 2023.PMID 36245074
- [6]Shakur H, Roberts I, Bautista R, et al. Effects of tranexamic acid on death, vascular occlusive events, and blood transfusion in trauma patients with significant haemorrhage (CRASH-2): a randomised, placebo-controlled trial Lancet (London, England), 2010.PMID 20554319
- [7]Borgman MA, Nishijima DK Tranexamic acid in pediatric hemorrhagic trauma The journal of trauma and acute care surgery, 2023.PMID 36044459
- [9]Neff LP, Beckwith MA, Russell RT, et al. Massive Transfusion in Pediatric Patients Clinics in laboratory medicine, 2021.PMID 33494884