Fellowship · Skin
Dermatology
Morphology, the systemic associations that change management, and the emergencies — every claim anchored to a primary source.
200Topics
1Chapters
189SAQs
198Cases
189Vivas
01Start here
Your first five minutes
- Step 1Read a verified topicStart with Atopic dermatitis — its clinical claims checked against the sources they cite.Open the topic
- Step 2Try five MCQsSingle best answer with worked explanations. Free while signed out.Practise now
- Step 3See what Pro addsThe complete library, every exam format, timed exams — one subscription.See pricing
02Begin here
Open these first
- 01Acne vulgarisAcne vulgaris is a chronic inflammatory disease of the pilosebaceous unit driven by follicular hyperkeratinisation, sebum overproduction, Cutibacterium acnes proliferation and innate immune activation. Fellowship-level assessment demands precise classification (comedonal, inflammatory, nodular, special forms), severity grading, differential diagnosis of acneiform eruptions, rational topical and systemic therapy with antibiotic stewardship, hormonal and isotretinoin regimens, management of acne fulminans, and awareness of scarring and psychosocial impact.Dermatology
- 02AlbinismAlbinism is a group of inherited disorders of melanin synthesis or melanosome biogenesis causing congenital hypopigmentation of skin, hair, and eyes (oculocutaneous albinism, OCA) or of the eyes alone (ocular albinism, OA1). The melanocytes are normal in NUMBER — the defect is in melanin PRODUCTION, which separates albinism from vitiligo (where melanocytes are lost). OCA1 (TYR, chr 11) is the severe tyrosinase-negative form; OCA2 (P gene, chr 15) is the most common worldwide and dominates in sub-Saharan Africa. All OCA types are autosomal recessive; OA1 (GPR143) is X-linked. Every type shares a characteristic OCULAR tetrad — nystagmus, foveal hypoplasia, iris transillumination, photophobia — plus abnormal chiasmal decussation. The dominant complication is SKIN CANCER (squamous cell carcinoma of the head and neck, up to 1000-fold risk), so lifelong sun protection (SPF 50+) and surveillance are the single most important interventions. Syndromic forms add systemic disease: Hermansky-Pudlak (platelet dense-granule deficiency, pulmonary fibrosis, colitis) and Chédiak-Higashi (neutrophil dysfunction, giant lysosomal granules, haemophagocytic lymphohistiocytosis).Dermatology
- 03Alopecia & Hair DisordersHair loss (alopecia) is the single most psychologically charged dermatological presentation. The pivotal skill is the scarring vs non-scarring distinction: non-scarring (reversible — androgenetic, alopecia areata, telogen effluvium, anagen effluvium, traction, trichotillomania, tinea capitis) preserves the follicle and is potentially recoverable, whereas scarring (cicatricial) (lichen planopilaris, frontal fibrosing alopecia, central centrifugal cicatricial alopecia, discoid lupus, folliculitis decalvans, pseudopelade) destroys follicular stem cells and is permanent and irreversible. Androgenetic alopecia (miniaturisation by dihydrotestosterone via 5-alpha-reductase type II) is graded by the Hamilton-Norwood scale in men and the Ludwig scale in women and treated with topical minoxidil 5% and oral finasteride 1 mg daily (men only). Alopecia areata (autoimmune T-cell attack on anagen follicles) presents as a smooth patch with exclamation-mark hairs and is now treated with JAK inhibitors (baricitinib, ritlecitinib) for severe disease — FDA-approved 2022-2023. Telogen effluvium is diffuse shedding two to three months after a trigger and is self-limiting.Dermatology
03Chapters
Curriculum map
1 chapters, 200 topics. Each card shows its topic count and how many of those are source-verified.
04Formats
How Derm is practised
- Topics200Browse topics for Derm.
- SAQs189Browse saqs for Derm.
- Cases198Browse cases for Derm.
- Vivas189Browse vivas for Derm.
PubMed-cited claims · MCQ, SAQ, case and viva formats · Educational exam preparation only — not medical advice.