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EM TopicsPelvic trauma

EM · Pelvic trauma

Pelvic trauma

The pelvic ring fracture and its life-threatening haemorrhage: the Young and Burgess classification, the pelvic binder at the greater trochanters, the FAST and the CT angiogram, the angiographic embolisation, the external fixation and the pelvic packing, the REBOA, and the associated injuries of the bladder, the urethra and the rectum.

high5 referencesUpdated 29 June 202619 min readVerification in progress

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ACEMFRCEMABEMFRCPCCCFPEMEBEEM

Red flags

  • The unstable pelvic fracture is a source of massive haemorrhage — the retroperitoneum can hold 3 to 4 litres of blood
  • The pelvic binder is applied at the level of the greater trochanters, not the iliac crests
  • A negative FAST does not exclude the pelvic bleeding — the blood is retroperitoneal
  • Blood at the urethral meatus with a pelvic fracture suggests a urethral injury — the catheter is withheld until the urethrogram
  • The AP compression and the vertical shear patterns bleed the most
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Practise this topic8 MCQs with explanations

Target exams

ACEMFRCEMABEMFRCPCCCFPEMEBEEM

Red flags

  • The unstable pelvic fracture is a source of massive haemorrhage — the retroperitoneum can hold 3 to 4 litres of blood
  • The pelvic binder is applied at the level of the greater trochanters, not the iliac crests
  • A negative FAST does not exclude the pelvic bleeding — the blood is retroperitoneal
  • Blood at the urethral meatus with a pelvic fracture suggests a urethral injury — the catheter is withheld until the urethrogram
  • The AP compression and the vertical shear patterns bleed the most

The pelvic fracture is one of the most dangerous injuries in the trauma, because the pelvis is a ring of bone surrounded by a rich venous plexus and the branches of the internal iliac artery, and the retroperitoneal space can hold 3 to 4 litres of blood before the tamponade. The unstable pelvic ring fracture — the open-book or the vertical shear — is the source of the massive haemorrhage that kills in the first hours, and its management is one of the few situations in which the emergency physician's intervention (the pelvic binder) directly saves the life. The pelvic fracture carries an overall mortality of 10 to 15 per cent, rising to 30 to 50 per cent in the open pelvic fracture and in the haemodynamically unstable patient, so the recognition of the unstable pattern and the immediate mechanical and the haematological resuscitation are the central competencies of the trauma team.[1]

The mechanism and the classification

The pelvic ring fracture is produced by the lateral compression (the side-impact collision, the crush), the anteroposterior compression (the open-book, the frontal collision), or the vertical shear (the fall from a height, the dashboard impact). The Young and Burgess classification classifies the patterns by the force vector and predicts the bleeding and the mortality. The lateral compression (LC) is the most common pattern (around 60 per cent of the pelvic fractures) and is generally the most stable; it is caused by the side-impact and produces the internal rotation and the crescent fracture of the sacrum, but the higher grades (the LC2 and the LC3) with the iliac wing or the sacral crushing can still bleed. The anteroposterior compression (APC) — the open-book — widens the pelvic volume, opens the sacroiliac joints and the symphysis pubis, and carries the highest transfusion requirement and a mortality of 20 to 30 per cent. The vertical shear (VS) — the Malgaigne — shears the hemipelvis vertically through the sacroiliac joint and the rami and is unstable in all the planes, with a mortality around 25 per cent. The combined mechanism (CM) is the fourth pattern and is the most lethal because it mixes the vectors and disrupts the ring in every direction.[1]

The Tile classification (the equivalent of the AO/OTA) classifies the biomechanical stability for the surgeon: the Type A is the stable, the avulsion and the isolated ramus; the Type B is the rotationally unstable but the vertically stable (the open-book and the lateral compression); the Type C is the unstable in all the planes (the vertical shear and the combined) and requires the surgical fixation. The Tile type guides the orthopaedic fixation, and the Young-Burgess type guides the emergency resuscitation because it predicts the bleeding. The two classifications are complementary and both are asked in the Fellowship examination.[1]

Tile A (stable)

  • The avulsion, the isolated ramus, the transverse sacral below S2
  • Stable in all planes — no ring disruption
  • No binder needed; the analgesia and the mobilisation
  • AO/OTA 61-A

Tile B (rotationally unstable, vertically stable)

  • The open-book (B1), the lateral compression (B2), the bilateral (B3)
  • The posterior sacroiliac ligaments intact — the vertical stability preserved
  • The binder and the external fixation reduce and stabilise
  • AO/OTA 61-B; the highest-yield binder responders

Tile C (unstable in all planes)

  • The vertical shear (C1), the bilateral (C2), the acetabular with the ring (C3)
  • The complete disruption of the posterior and the sacroiliac complex
  • Requires the surgical fixation; the highest mortality and transfusion
  • AO/OTA 61-C; the angiographic and the packing candidates

LC (lateral compression)

  • Around 60 per cent — the commonest pattern; the side-impact and the crush
  • The internal rotation; the crescent sacral fracture; the rami overlap
  • Generally stable (LC1); the LC2/LC3 bleed with the iliac wing or the sacral crush
  • The binder less beneficial — may over-reduce

APC (open-book)

  • The frontal collision, the crush from the front; the external rotation
  • The symphysis and the sacroiliac widening; the increased pelvic volume
  • The highest transfusion requirement; the mortality of 20 to 30 per cent
  • The binder is the first-line and the most effective

VS (vertical shear)

  • The fall from the height, the dashboard impact; the cranial displacement
  • The hemipelvis sheared vertically through the SI joint and the rami
  • Unstable in all planes (Tile C); the mortality around 25 per cent
  • The binder plus the skeletal traction for the displaced hemipelvis

CM (combined mechanism)

  • The mix of the vectors; the ring disrupted in every direction
  • The most lethal pattern; the polytrauma and the head injury common
  • Demands the immediate binder, the blood and the multidisciplinary escalation
  • The highest mortality of all the Young-Burgess patterns
The venous plexus, not the artery, is the source of most of the bleedingThe posterior pelvic venous plexus and the cancellous bone surfaces account for 80 to 85 per cent of the pelvic bleeding; the arterial bleeding from the branches of the internal iliac (the superior gluteal, obturator, internal pudendal) accounts for only 10 to 20 per cent. This is why the binder — which tamponades the venous and the bony bleeding by reducing the volume — works at all, and why the angiographic embolisation is reserved for the persistent arterial blush: the embolisation addresses only the minority of the bleeding, and the binder addresses the majority.[1]
The Tile type guides the operating room; the Young-Burgess type guides the resus bayThe two classifications answer different questions. The Tile/AO type answers the question of the surgeon — is the ring stable enough to fix, and how? The Type C needs the open or the percutaneous fixation. The Young-Burgess type answers the question of the emergency physician — how much will this bleed, and does the binder help? The APC and the vertical shear bleed the most and respond best to the binder; the lateral compression bleeds least and the binder may over-reduce it. The Fellowship candidate is expected to use both, in the same sentence.[1]
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References5ShowHide
  1. [1]Cullinane DC, Schiller HJ, Zielinski MD, et al. Eastern Association for the Surgery of Trauma practice management guidelines for hemorrhage in pelvic fracture--update and systematic review J Trauma, 2011.PMID 22182895
  2. [2]Rossaint R, Bouillon B, Cerny V, et al. The European guideline on management of major bleeding and coagulopathy following trauma: sixth edition Crit Care, 2023.PMID 36859355
  3. [3]Shakur H, Roberts I, Bautista R, et al. Effects of tranexamic acid on death, vascular occlusive events, and blood transfusion in trauma patients with significant haemorrhage (CRASH-2): a randomised, placebo-controlled trial Lancet, 2010.PMID 20554319
  4. [4]Holcomb JB, Tilley BC, Baraniuk S, et al. Transfusion of plasma, platelets, and red blood cells in a 1:1:1 vs a 1:1:2 ratio and mortality in patients with severe trauma: the PROPPR randomized clinical trial JAMA, 2015.PMID 25647203
  5. [5]Morrison JJ, Dubose JJ, Rasmussen TE, Midwinter MJ. Military Application of Tranexamic Acid in Trauma Emergency Resuscitation (MATTERs) Study Arch Surg, 2012.PMID 22006852

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