Paeds Vivas · paediatric-dermatology
Psoriasis in children — branching viva
Branching structured-oral viva on psoriasis in children: the chronic plaque and guttate forms and the paediatric-specific face, scalp, nail and napkin presentations; the IL-23 and Th17 and IL-17 pathophysiology with HLA-Cw6 genetics and streptococcal and Koebner triggers; the differential from atopic and seborrhoeic dermatitis and tinea; clinical diagnosis with severity measured by body surface area and CDLQI; stepwise treatment from emollients and a potency-matched topical corticosteroid plus calcipotriol through narrowband UVB to methotrexate, cyclosporin, acitretin and the biologics; and the obesity, juvenile psoriatic arthritis and psychosocial comorbidities.
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Study tools
Target exams
Opening question
Examiner: Take me through this child. What is the diagnosis, and what is your frame for managing it? [1]
Candidate: The most likely diagnosis is chronic plaque psoriasis — the thick, well-demarcated, scaly patches on the extensor elbows, knees and scalp, with nail pitting and a father with psoriasis, are the classic picture of the commonest form of childhood psoriasis. My frame is three-layered: match the treatment to the severity and quality-of-life impact, screen the comorbidities that travel with psoriasis, and build a long-term partnership with the family because this is a chronic relapsing disease. Because she is otherwise well with limited sites, this begins with topical therapy and a comorbidity screen, with escalation if the disease or its impact grows. [1] [2]
Examiner: How do you classify paediatric psoriasis, and how does it differ from the adult disease? [2]
Candidate: By lesion shape and site. Chronic plaque psoriasis is the commonest — well-demarcated erythematous plaques with silvery-white scale on the extensor surfaces and scalp, with the Auspitz sign of pinpoint bleeding and Koebner phenomenon where plaques arise in lines of trauma. Acute guttate psoriasis is the other classic form — small droplet papules across the trunk one to two weeks after streptococcal pharyngitis. Children differ from adults: the face is involved more often, the scalp is a frequent first site, and infants can present with napkin or diaper-area psoriasis. Nail and scalp involvement are common, and I would examine all of these. [2] [3]
References7ShowHide
- [1]Menter A; Cordoro KM; Davis DMR; Feldman SR; et al Joint American Academy of Dermatology-National Psoriasis Foundation guidelines of care for the management and treatment of psoriasis in pediatric patients. J Am Acad Dermatol, 2020.PMID 31703821
- [2]Eichenfield LF; Paller AS; Tom WL; Lara-Corrales I; et al Pediatric psoriasis: Evolving perspectives. Pediatr Dermatol, 2018.PMID 29314219
- [3]Mahé E Childhood psoriasis. Eur J Dermatol, 2016.PMID 27900946
- [7]Maruani A; Samimi M; Stembridge N; McBride S; et al Non-antistreptococcal interventions for acute guttate psoriasis or an acute guttate flare of chronic psoriasis. Cochrane Database Syst Rev, 2019.PMID 30958563
- [9]Paller AS; Siegfried EC; Langley RG; Gottlieb AB; et al Etanercept treatment for children and adolescents with plaque psoriasis. N Engl J Med, 2008.PMID 18199863
- [10]Aljalfan AA; Maniya MT; Sachedina D; Alavi A; et al Biologics for treatment of paediatric plaque psoriasis: A systematic review and network meta-analysis. J Eur Acad Dermatol Venereol, 2026.PMID 41090545
- [11]Phan K; Lee G; Fischer G Pediatric psoriasis and association with cardiovascular and metabolic comorbidities: Systematic review and meta-analysis. Pediatr Dermatol, 2020.PMID 32436322