ICU · Respiratory
Pulmonary contusion and blunt chest trauma
Also known as Pulmonary contusion · Blunt chest injury · Flail chest · Blunt aortic injury
Pulmonary contusion is bruising of the lung parenchyma from blunt chest trauma (motor vehicle crash, fall, crush, blast). Alveolar-capillary disruption → alveolar haemorrhage + interstitial/peribronchial oedema → impaired gas exchange (V/Q mismatch → true shunt) + reduced compliance → progressive hypoxaemia that develops and WORSENS over 24-48h (worsens before improving). Often associated with rib fractures (1 association), flail chest (≥3 consecutive ribs fractured in ≥2 places — paradoxical breathing), pneumothorax, haemothorax, blunt aortic injury and cardiac (myocardial) contusion. Management: oxygen (escalate via HFNC/NIV), analgesia (epidural or thoracic paravertebral block for rib fractures), JUDICIOUS fluids (avoid overload — worsens contusion), NIV if moderate respiratory distress, intubation + lung-protective ventilation (Vt 6 mL/kg PBW) if severe. Monitor for: ARDS (develops in ~20% of severe contusions), pneumonia, respiratory failure — mortality 10-25%. CXR may be NORMAL initially (lags 24-48h); CT chest is more sensitive and quantifies contusion volume.
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Red flags
- Pulmonary contusion WORSENS over 24-48h — CXR may be normal initially, worsens over hours. Don't be reassured by an early normal film
- Flail chest: ≥3 consecutive ribs fractured in ≥2 places → paradoxical chest wall movement — requires analgesia (epidural) + may need NIV/ventilation
- Blunt aortic injury: widened mediastinum on CXR — CT angiography to confirm. Life-threatening
- Excessive IV fluids WORSEN pulmonary contusion (capillary leak + hydrostatic pressure → more oedema). Conservative/dry strategy
- Hypoxaemia may be delayed — observe 24-48h even if the patient looks well initially
- ≥3 rib fractures in age >65 carries 10-20% mortality — admit to HDU/ICU, aggressive analgesia
Meet the patient
A 72-year-old after a high-speed collision has bruising, six rib fractures and a normal initial chest radiograph. Six hours later he is tachypnoeic and hypoxaemic, with increasing work of breathing despite oxygen.[1][2]
The early film was not the diagnosis. The trajectory is the clue: contusion evolves, pain splints ventilation, and an apparently well patient can deteriorate during the next day.[1][2]
| Injury | Mechanism | Hallmark features | First-line management |
|---|---|---|---|
| Pulmonary contusion | Direct impact → alveolar-capillary disruption | Hypoxaemia WORSENS over 24-48h; patchy infiltrates under the injury; CXR lags CT | Oxygen, NIV, judicious fluids, lung-protective ventilation if intubated |
| Flail chest | ≥3 consecutive ribs fractured in ≥2 places | Paradoxical chest wall movement; severe pain; respiratory failure | Aggressive analgesia (epidural), NIV, surgical rib fixation (selected) |
| Blunt cardiac injury | Direct impact, heart crushed between sternum and spine | Troponin elevated, arrhythmia (AF, PVCs, heart block), ECG changes | ECG monitoring 24-48h, troponin, echo, treat arrhythmia |
| Traumatic aortic injury | Deceleration → intimal tear / partial transection | Widened mediastinum, apical cap, BP differential | CT angiography → TEVAR (preferred) or open repair |
| Tension pneumothorax | Air under pressure, one-way valve | Hypotension, tracheal deviation, absent breath sounds, distended neck veins | IMMEDIATE needle decompression (don't wait for CXR), then chest drain |
| Haemothorax | Bleeding into pleural space | Dullness, decreased breath sounds, hypovolaemia | Large-bore chest drain (28-36 Fr); thoracotomy if >1500 mL or >200 mL/h |
| Tracheobronchial injury | Crush/deceleration, tears at carina | Massive continuous air leak, non-re-expanding pneumothorax, surgical emphysema | Bronchoscopy, surgical repair, double-lumen tube |
| Diaphragmatic injury | Penetrating thoracoabdominal or blunt rupture (L>R) | Delayed herniation; bowel sounds in chest; NG tube curled in chest | Surgical repair (laparotomy/thoracotomy) |
References22ShowHide
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