ICU · Cardiovascular
Cardiac arrhythmia management in the ICU
Also known as Atrial fibrillation · Ventricular tachycardia · Bradycardia and heart block · Cardiac arrhythmia · Supraventricular tachycardia · Atrial flutter · Torsades de Pointes · Complete heart block · Ventricular fibrillation · Antiarrhythmic pharmacology
Arrhythmias are common in ICU (up to 20% of patients). Types: atrial fibrillation (1 — new-onset AF in ICU is associated with worse outcomes), ventricular tachycardia/fibrillation (life-threatening — usually cardiac origin), bradyarrhythmias (heart block, sinus bradycardia), supraventricular tachycardia (AVNRT/AVRT), atrial flutter. Management principles: (1) is the patient haemodynamically unstable? → cardiovert/defibrillate immediately. (2) Identify and treat underlying cause (electrolytes, ischaemia, sepsis, drugs). (3) Rate vs rhythm control. AF: rate control (beta-blocker, diltiazem) usually first; rhythm control (amiodarone, DC cardioversion) if unstable. VT/VF: defibrillate + amiodarone. Bradycardia: atropine → adrenaline/pacing. SVT: vagal manoeuvres → adenosine 6→12→12 mg. Torsades: IV magnesium, stop QT-prolonging drugs, overdrive pacing.
Checked against its sources on 2 Sept 2026
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Target exams
Red flags
- Haemodynamic instability + arrhythmia = IMMEDIATE cardioversion/defibrillation (do NOT wait for drugs)
- Check potassium and magnesium FIRST in any arrhythmia — correct before antiarrhythmics
- New-onset AF in ICU: often resolves once underlying cause treated (sepsis, hypoxia, electrolytes)
- Torsades de Pointes: check QT, stop causative drugs, IV magnesium sulphate 2 g even if Mg normal
- Do NOT give adenosine in broad-complex tachycardia (may be VT — adenosine can cause VF) or atrial flutter with pre-excited conduction (WPW)
- Avoid calcium channel blockers (verapamil/diltiazem) in heart failure with reduced EF — negative inotrope, may precipitate arrest
- Complete heart block with ventricular escape needs transvenous pacing — transcutaneous is only a bridge
Meet the patient
A 68-year-old man is admitted to ICU with septic shock from a urinary source — intubated, on noradrenaline 0.3 mcg/kg/min, BP 95/60, lactate 3.2. At 3am the monitor shows a new irregularly irregular rhythm at 150, and the blood gas comes back K+ 3.3, Mg2+ 0.6. He has no cardiac history.[2] [4]
The two questions that will decide his next hour are the two that decide every ICU arrhythmia: is he haemodynamically unstable? (he is not yet — BP held by noradrenaline, no ischaemia, no failure) and what provoked this? (sepsis, catecholamines, and a low potassium — exactly the triad that drives new-onset AF in the ventilated septic patient). Hold those two questions and the rate-versus-rhythm choice answers itself.[3]
References33ShowHide
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