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EM VivasTachyarrhythmias

EM Vivas · Tachyarrhythmias

Tachyarrhythmias — the structured approach and the dangerous traps

A clinical viva on the tachycardia algorithm and its pitfalls.

clinical2 min readSource-verified ·

Target exams

ACEMFRCEMABEM
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Target exams

ACEMFRCEMABEM
Prompt
Describe your structured approach to the patient who presents to the emergency department with a tachyarrhythmia, and explain the dangerous traps and their avoidance.

Interpretation

Reveal interpretationShowHide

The structured approach:

  1. Stability first: the unstable tachyarrhythmia (the shock, the ischaemic chest pain, the acute heart failure, the altered consciousness) receives the synchronised DC cardioversion, regardless of the rhythm.
  2. QRS width: the narrow (below 120 ms) is the supraventricular; the broad (above 120 ms) is the ventricular or the aberrant.
  3. Regularity: within each, the regular versus the irregular narrows the differential.
  4. The specific rhythm and the drug follow.

The narrow-regular: the AVNRT and the AVRT — the vagal manoeuvres (the modified Valsalva) then the IV adenosine 6-12-12 mg.[3] The atrial flutter at around 150 — the adenosine unmasks, the cardioversion or the rate control terminates.

The narrow-irregular: the atrial fibrillation — the rate versus the rhythm control, the 48-hour rule (the rhythm control reasonable under 48 hours; the rate control and the anticoagulation beyond, with the three-week anticoagulation or the TOE before the cardioversion), and the CHA2DS2-VASc for the long-term anticoagulation.[4]

The broad-regular: the ventricular tachycardia — the IV amiodarone (stable) or the cardioversion (unstable). Treat as VT if uncertain.[2]

The broad-irregular: the torsades (the IV magnesium bolus, the potassium, withdraw drug) and the atrial fibrillation with the Wolff-Parkinson-White (no AV-node blockers; cardiovert or a pathway drug).[2][3]

The dangerous traps:

  • The verapamil or the adenosine in the broad-complex tachycardia (treat as the VT — the verapamil kills in the VT).[2]
  • The AV-node blocker in the atrial fibrillation with the WPW (the varying QRS, the irregular broad — the blocker diverts the conduction down the accessory pathway, the VF).[3]
  • The adenosine in the asthma (avoid — the bronchospasm).[3]
  • The cardioversion of the AF beyond 48 hours without the anticoagulation or the TOE (the stroke).[4]
  • The unsynchronised shock on the cardioversion (the VF).[3]
[2]
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References3ShowHide
  1. [2]Kashou AH, Noseworthy PA, DeSimone CV, et al. Wide complex tachycardia differentiation: a reappraisal of the state-of-the-art. Journal of the American Heart Association, 2020.PMID 32427020
  2. [3]Brugada J, Katritsis DG, Arbelo E, et al. 2019 ESC Guidelines for the management of patients with supraventricular tachycardiaThe Task Force for the management of patients with supraventricular tachycardia of the European Society of Cardiology (ESC) Eur Heart J, 2020.PMID 31504425
  3. [4]Joglar JA, Chung MK, Armbruster AL, et al. 2023 ACC/AHA/ACCP/HRS Guideline for the Diagnosis and Management of Atrial Fibrillation: A Report of the American College of Cardiology/American Heart Association Joint Committee on Clinical Practice Guidelines Circulation, 2024.PMID 38033089
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