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Anaes TopicsAirway management

Anaes · Airway management

Airway emergencies: recognition and the structured response

Also known as Airway emergencies · The airway crisis · The lost airway · Intraoperative airway emergency · Acute airway obstruction · Crisis resource management in the airway

An airway emergency is any acute failure to oxygenate or ventilate — from laryngospasm and bronchospasm to aspiration, obstruction, the lost airway and cannot-intubate-cannot-oxygenate. Survival hinges less on a single heroic technique than on early recognition, calling for help, basic airway manoeuvres applied well, and a structured, rehearsed escalation — because the time from a falling saturation to a hypoxic cardiac arrest is short.

high8 referencesUpdated 26 July 20267 min readVerification in progress

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Red flags

  • A falling saturation with failing ventilation or obstruction is an airway emergency until proven otherwise — the margin to hypoxic cardiac arrest is measured in minutes.
  • Hypoxic bradycardia in an airway crisis signals imminent arrest; the treatment is oxygenation by whatever means, not an antiarrhythmic — atropine alone does not prevent arrest from ongoing hypoxia.
  • Inspiratory stridor, tracheal tug and recession indicate partial obstruction that may become complete; preserve spontaneous ventilation and call for senior help.
  • Failure of mask ventilation AND a supraglottic airway is cannot-intubate-cannot-oxygenate until the airway is recovered — declare it and move to front-of-neck access without delay.
  • Repeated laryngoscopy attempts during a crisis worsen bleeding, oedema and physiological decompensation; cap attempts and escalate.
  • Unrecognised oesophageal intubation is fatal — confirm every tube placement with waveform capnography, especially in the emergency.
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Your progress

Saved on this device.

Practise this topic8 MCQs with explanations

Target exams

ANZCAFRCAABAEDAICFCAIFCA_SA

Red flags

  • A falling saturation with failing ventilation or obstruction is an airway emergency until proven otherwise — the margin to hypoxic cardiac arrest is measured in minutes.
  • Hypoxic bradycardia in an airway crisis signals imminent arrest; the treatment is oxygenation by whatever means, not an antiarrhythmic — atropine alone does not prevent arrest from ongoing hypoxia.
  • Inspiratory stridor, tracheal tug and recession indicate partial obstruction that may become complete; preserve spontaneous ventilation and call for senior help.
  • Failure of mask ventilation AND a supraglottic airway is cannot-intubate-cannot-oxygenate until the airway is recovered — declare it and move to front-of-neck access without delay.
  • Repeated laryngoscopy attempts during a crisis worsen bleeding, oedema and physiological decompensation; cap attempts and escalate.
  • Unrecognised oesophageal intubation is fatal — confirm every tube placement with waveform capnography, especially in the emergency.
The one-line answer

An airway emergency is any acute failure to oxygenate or ventilate — from laryngospasm and bronchospasm to aspiration, obstruction, the lost airway, and cannot-intubate-cannot-oxygenate. Survival hinges less on any single device than on early recognition, an immediate call for help, basic airway manoeuvres applied well, and a structured, rehearsed escalation — because the time from a falling saturation to a hypoxic cardiac arrest is short.[6][7]

Meet the patient

A fit twenty-year-old emerges from a brief general anaesthetic for a hand surgery. He coughs on the tube, the cords snap shut, and within thirty seconds the saturation is in the eighties and the heart rate is slowing. The monitor's bradycardia alarm is the heart surrendering to hypoxia, not a primary rhythm problem.[1][6]

The decision that saves this patient: do not chase the bradycardia with an antimuscarinic — chase the hypoxia with oxygenation. Everything in this topic hangs off that discipline — recognise early, call early, oxygenate by whatever means works, and escalate through a rehearsed sequence before the saturation reaches the range from which recovery is unlikely.[1]

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References8ShowHide
  1. [1]Pařízek T, Waldauf P, Kalina M, et al. Effect of atropine on time to cardiac arrest in hypoxic bradycardia: a randomised cross-over study in a porcine apnoea model BJA Open, 2026.PMID 42317406
  2. [2]Wu X, et al. Airway Decision-Making in Acute Epiglottitis: A Case Report of Planned Tracheostomy in a Can Ventilate but Cannot Intubate (CVCI) Scenario Clin Case Rep, 2026.PMID 42290811
  3. [3]Mullally ME, et al. Evaluation of novel materials for front-of-neck access simulations Anaesth Intensive Care, 2026.PMID 42290043
  4. [4]Sato M, et al. Suspected Neostigmine-Associated Bronchospasm Complicated by Pulmonary Edema During General Anesthesia: A Case Report Cureus, 2026.PMID 42311723
  5. [5]Del Tedesco JC, et al. [The mindset of anesthesiologists while performing surgical airway management]. Anaesthesiologie, 2026.PMID 42230350
  6. [6]Karim HMR, et al. Letter to the Editor: Insight into emergency endotracheal intubation in critically ill and strategies beyond equipment World J Crit Care Med, 2026.PMID 42272892
  7. [7]Frerk C, Mitchell VS, McNarry AF, et al. Difficult Airway Society 2015 guidelines for management of unanticipated difficult intubation in adults Br J Anaesth, 2015.PMID 26556848
  8. [8]Al-alami AA, Zestos MM, Baraka AS. Pediatric laryngospasm: prevention and treatment Curr Opin Anaesthesiol, 2009.PMID 19434787

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