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LibraryNephrology / General Medicine

MBBS OSCE · Nephrology / General Medicine

OSCE — assessment and emergency management of acute kidney injury with hyperkalaemia

An 8-minute OSCE station assessing the candidate's structured assessment of AKI — volume status, classification (pre-/intrinsic/post-renal), FENa interpretation, KDIGO staging, the hyperkalaemia emergency ladder, and the AEIOU dialysis indications.

8 min stationSource-verified ·

Exam tags

NEET-PGINICET
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Exam tags

NEET-PGINICET

Brief (to candidate)

A 68-year-old diabetic woman is admitted with 3 days of diarrhoea and vomiting; she has been taking ibuprofen for back pain. She is drowsy,[1] pulse 110, BP 88/56, JVP low, with a dry mouth and reduced skin turgor. Bloods show creatinine 3.0 mg/dL (baseline 1.0), potassium 6.9 mmol/L, venous pH 7.18; the ECG shows wide peaked T waves. Urine output is 0.15 mL/kg/h. You have 8 minutes to assess her, classify and stage the AKI, and outline the immediate management.

Candidate instructions

  1. Take a focused history and examine using an ABCDE approach, paying attention to volume status and any obstructive signs.[1]
  2. Classify the AKI (pre-/intrinsic/post-renal) and state the KDIGO stage.
  3. Outline the immediate management, including the hyperkalaemia ladder with doses and the indications for renal replacement therapy.
  4. State your disposition (ward vs ICU) and the safety-net / follow-up advice.[3]

Examiner checklist (mark each domain / 10)

DomainKey actions expected
ABCDE / volume status[1]Recognises hypovolaemia (tachycardia, hypotension, low JVP, dry mucosae); plans a fluid challenge 250-500 mL balanced crystalloid with reassessment
Hyperkalaemia emergencyIdentifies K 6.9 with ECG changes as the immediate threat; gives calcium gluconate 10% 10 mL IV first; then insulin 10 units + 50% dextrose
ClassificationPre-renal (volume depletion + NSAID); excludes obstruction (history, examination, ultrasound)
KDIGO stagingReproduces staging; creatinine 3.0x baseline = Stage 3 (worst criterion)
Stop nephrotoxins / drug reviewStops the NSAID; reviews all drugs for renal dosing
AEIOUReproduces the indications (acidosis, electrolytes, ingestion, overload, uraemia); states creatinine alone is NOT an indication
Communication & safety-netClear plan; ICU if shock/AEIOU; nephrology if intrinsic/rising; follow-up renal function (risk of future CKD)

Model key actions

  • Calcium gluconate 10% 10 mL IV for the hyperkalaemic ECG changes (stabilises the myocardium; does not lower potassium).[3]
  • Fluid challenge (250-500 mL balanced crystalloid) to restore perfusion; stop the NSAID; treat the GI losses.[1]
  • Classify pre-renal (FENa under 1% expected); KDIGO Stage 3 (creatinine 3.0x baseline).[1]
  • AEIOU drives dialysis — this patient's hyperkalaemia should resolve medically; dialyse only if refractory.[2]
  • Refer to nephrology/ICU for shock or an AEIOU indication; follow up renal function — AKI survivors risk future CKD.[3]

Common errors

  • Treating the creatinine before the potassium — the ECG changes make hyperkalaemia the lethal, immediate problem.
  • Not recognising volume depletion — failing to give a fluid challenge in a clearly pre-renal AKI.
  • Continuing the NSAID or other nephrotoxins.
  • Dialysing for a creatinine number rather than an AEIOU indication.
  • Missing obstruction — not requesting an ultrasound or checking bladder/urinary symptoms.[3]
References3ShowHide
  1. [1]Kellum JA, Lameire N Diagnosis, evaluation, and management of acute kidney injury: a KDIGO summary (Part 1). Critical Care, 2013.PMID 23394211
  2. [2]Lameire N, Kellum JA Contrast-induced acute kidney injury and renal support for acute kidney injury: a KDIGO summary (Part 2). Critical Care, 2013.PMID 23394215
  3. [3]Ronco C, Bellomo R, Kellum JA. Acute kidney injury. Lancet, 2019.PMID 31777389