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Psych MEQs / SAQsChild and adolescent psychiatry — neurodevelopmental

Psych MEQs / SAQs · Child and adolescent psychiatry — neurodevelopmental

ADHD across the lifespan — assessment and multimodal management (MEQ)

FRANZCP-style modified essay on school-age ADHD: multi-informant assessment, differentials, stimulant and atomoxetine/guanfacine options with monitoring, MTA logic, SUD and future driving counselling.

20 marks20 min2 min readVerification in progress

Target exams

FRANZCPMRCPsychABPNMD-DNB
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Target exams

FRANZCPMRCPsychABPNMD-DNB
Prompt
A 10-year-old boy is referred by his school with incomplete work, impulsive calling out, and playground risk-taking. Parents report similar difficulties since early primary school at home and with grandparents. Teacher Conners and parent scales are elevated. He has no known cardiac disease. There is a family history of ADHD in his father. He has mild oppositional behaviour but no clear manic episodes. He is beginning to experiment with his older brother's energy drinks. (i) Outline assessment priorities including multi-informant evidence, differentials and baseline work-up before medication. (ii) State working diagnosis and key discriminators from anxiety, learning disorder and bipolar spectrum. (iii) Outline a multimodal plan including a named first-line stimulant approach with monitoring, and an alternative non-stimulant with approximate dosing framework. (iv) Explain how MTA findings inform the relative roles of medication and behavioural treatment. (v) List counselling points on substance risk and, looking ahead, driving. (20 marks)

Model answer

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(i) Assessment priorities. Multi-informant history (child, parents, school) covering inattention and hyperactivity-impulsivity symptoms, onset before age 12, multi-setting presence, and functional impairment. Review report cards/prior assessments. Screen comorbidity: ODD, learning disorder, anxiety, mood, tics, sleep, substances (energy drinks as gateway curiosity). Developmental and family history (father ADHD). Baseline before medication: height, weight, BP, HR, cardiac history and family sudden death; consider ECG if indicated by history/exam. Hearing/vision and sleep history. Consider psychoeducational testing if learning disorder suspected. Shared decision-making and consent with parents and developmentally with the child. Local prescribing authority requirements for stimulants.[4]

(ii) Working diagnosis and differentials. Working diagnosis: ADHD combined presentation, moderate-severe, with mild oppositional features; rule out/formulate learning disorder contribution. Anxiety: worry-linked inattention without lifelong multi-setting developmental pattern. Learning disorder: skill-specific underachievement with relatively preserved attention elsewhere. Bipolar: episodic elevated mood, decreased need for sleep, grandiosity — not chronic from early school years. Substance-induced symptoms: currently low-level; still counsel. Medical mimics only if red flags (sleep apnoea, thyroid, hearing).[4]

(iii) Multimodal plan. Psychoeducation; behavioural parent training; classroom strategies (seating, chunked tasks, daily report card). For moderate-severe impairment, first-line stimulant example: long-acting methylphenidate product-specific morning dosing with weekly titration to response/tolerability (IR alternatives if needed), monitoring height/weight, BP, HR, appetite, sleep, mood/tics, adherence and school function. Alternative non-stimulant: atomoxetine start ~0.5 mg/kg/day then toward ~1.2 mg/kg/day (max ~1.4 mg/kg or 100 mg per many labels) with delayed onset counselling; or guanfacine XR from 1 mg with BP/HR/sedation monitoring if preferred/adjunct pathway. Review response with rating scales and teacher feedback.[2][3][4]

(iv) MTA logic. At 14 months, carefully managed medication and combined treatment were superior to behavioural treatment alone and community care for core ADHD symptoms; combination may help non-ADHD domains. Behavioural interventions remain important for broader function and parent skills even when medication is used.[1]

(v) Substance and driving counselling. ADHD elevates lifetime SUD risk; early open discussion about diversion, peers, cannabis/alcohol, and safe storage of stimulants. Looking ahead to adolescence: elevated relative road traffic risk; counsel distraction/impulsivity, sleep, substances, medication timing; document advice; licensing/reporting is jurisdiction-specific.[5][6]

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References6ShowHide
  1. [1]The MTA Cooperative Group A 14-month randomized clinical trial of treatment strategies for attention-deficit/hyperactivity disorder Arch Gen Psychiatry, 1999.PMID 10591283
  2. [2]Cortese S, Adamo N, Del Giovane C, et al. Comparative efficacy and tolerability of medications for attention-deficit hyperactivity disorder in children, adolescents, and adults: a systematic review and network meta-analysis Lancet Psychiatry, 2018.PMID 30097390
  3. [3]Michelson D, Allen AJ, Busner J, et al. Once-daily atomoxetine treatment for children and adolescents with attention deficit hyperactivity disorder: a randomized, placebo-controlled study Am J Psychiatry, 2002.PMID 12411225
  4. [4]Pliszka S; AACAP Work Group on Quality Issues Practice parameter for the assessment and treatment of children and adolescents with attention-deficit/hyperactivity disorder J Am Acad Child Adolesc Psychiatry, 2007.PMID 17581453
  5. [5]Wilens TE Attention deficit hyperactivity disorder and substance use disorders Am J Psychiatry, 2006.PMID 17151154
  6. [6]Vaa T ADHD and relative risk of accidents in road traffic: a meta-analysis Accid Anal Prev, 2014.PMID 24238842
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