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Paeds Vivasgenetics-dysmorphology-and-metabolism

Paeds Vivas · genetics-dysmorphology-and-metabolism

Williams syndrome — branching viva

Branching viva on Williams syndrome: recognising the multisystem pattern of the 7q11.23 microdeletion, explaining the ELN-driven elastin arteriopathy and coronary risk, confirming the deletion with chromosomal microarray, managing infantile hypercalcaemia, and counselling the autosomal dominant inheritance.

branching clinical structured oral3 min readVerification in progress

Target exams

RACP DCEMRCPCH ClinicalRCPSC Pediatrics
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Study tools

Target exams

RACP DCEMRCPCH ClinicalRCPSC Pediatrics
Prompt
Outpatient clinic: an eight-month-old infant with failure to thrive, irritability, a distinctive facies with periorbital fullness and a stellate iris, a loud ejection systolic murmur radiating to the neck, and elevated four-limb blood pressure. The examiner asks: what is the unifying diagnosis and the chromosomal abnormality, which gene drives the cardiovascular phenotype, why does haploinsufficiency produce stenotic arteries, what is the first-line confirmatory test, and what is the cardiac surveillance obligation — then branches to the finding of hypercalcaemia and asks how you manage it, and finally to the question of whether the deletion is inherited and what that means for the family.

Opening framework

My framework has four layers. First, the pattern — a dysmorphic infant with a murmur, hypertension, and metabolic disturbance points to a contiguous-gene microdeletion syndrome. Second, the gene — the cardiovascular phenotype is driven by ELN haploinsufficiency, producing an elastin arteriopathy. Third, the test — a chromosomal microarray confirms the 7q11.23 deletion. Fourth, the family — the deletion is autosomal dominant, so a parent who carries it confers a 50 percent recurrence risk. [1] [2]

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References6ShowHide
  1. [1]Morris CA, et al. Health Care Supervision for Children With Williams Syndrome. Pediatrics, 2020.PMID 31964759
  2. [2]Kozel BA, et al. Williams syndrome. Nat Rev Dis Primers, 2021.PMID 34140529
  3. [3]Collins RT 2nd. Cardiovascular disease in Williams syndrome. Curr Opin Pediatr, 2018.PMID 30045083
  4. [5]Merla G, et al. Supravalvular aortic stenosis: elastin arteriopathy. Circ Cardiovasc Genet, 2012.PMID 23250899
  5. [6]Sindhar S, et al. Hypercalcemia in Patients with Williams-Beuren Syndrome. J Pediatr, 2016.PMID 27574996
  6. [7]Twite MD, Stenquist S, Ing RJ Williams syndrome. Paediatr Anaesth, 2019.PMID 30811742
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