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Paeds Vivascardiology

Paeds Vivas · cardiology

Ventricular septal defect — branching viva

Branching viva from the recognition of the VSD murmur through the anatomic and haemodynamic classification, the four-to-eight-week heart-failure physiology, the device-versus-surgery decision, and the Eisenmenger management with family and pregnancy counselling.

branching clinical structured oral3 min readVerification in progress

Target exams

RACP General PaediatricsRACP DCEMRCPCH ClinicalRCPSC Pediatrics
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Study tools

Target exams

RACP General PaediatricsRACP DCEMRCPCH ClinicalRCPSC Pediatrics
Prompt
You are the paediatric registrar in clinic. The consultant asks you to talk through four children: a well six-week-old with a loud murmur found at the check, a three-month-old in overcirculation failure, a two-year-old with a perimembranous VSD and new aortic regurgitation, and a sixteen-year-old lost to follow-up who now has Eisenmenger physiology.

Station opening

Examiner: "Define the ventricular septal defect and explain why its clinical course is set by its size." [1]

Strong candidate (must-hit)

  • Defines the VSD as a congenital communication between the ventricles producing a left-to-right shunt because the left ventricle pumps at higher pressure; explains that the clinical course is set by shunt size, which depends on the defect size and the pulmonary vascular resistance, running from a tiny defect that closes to a large defect that floods the lungs, causes heart failure, and over years risks Eisenmenger syndrome. [1]

Weak candidate

  • "A VSD is a hole in the heart that some children are born with." [1]
Paediatrics Fellowship Pro

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You have read the opening of this viva. The complete unit — every section and its primary-source references — is part of the Paediatrics Fellowship fellowship library.

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References7ShowHide
  1. [1]Hoffman JI; Kaplan S The incidence of congenital heart disease. J Am Coll Cardiol, 2002.PMID 12084585
  2. [3]Du ZD; Roguin N; Wu XJ Spontaneous closure of muscular ventricular septal defect identified by echocardiography in neonates. Cardiol Young, 1998.PMID 9855105
  3. [4]Butera G; Carminati M; Chessa M; et al Transcatheter closure of perimembranous ventricular septal defects: early and long-term results. J Am Coll Cardiol, 2007.PMID 17868812
  4. [6]Galiè N; Beghetti M; Gatzoulis MA; Granton J; et al Bosentan therapy in patients with Eisenmenger syndrome: a multicenter, double-blind, randomized, placebo-controlled study. Circulation, 2006.PMID 16801459
  5. [7]Gatzoulis MA; Beghetti M; Galiè N; Granton J; et al Longer-term bosentan therapy improves functional capacity in Eisenmenger syndrome: results of the BREATHE-5 open-label extension study. Int J Cardiol, 2008.PMID 17658633
  6. [8]Wilson WR; Gewitz M; Lockhart PB; Bolger AF; et al Prevention of Viridans Group Streptococcal Infective Endocarditis: AHA Scientific Statement. Circulation, 2021.PMID 33853363
  7. [9]Baumgartner H; De Backer J The ESC Clinical Practice Guidelines for the Management of Adult Congenital Heart Disease. Eur Heart J, 2020.PMID 33128054
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