Paeds Vivas · acute-care-resuscitation-and-toxicology
Fluid bolus therapy and vasoactive support — branching viva
A branching viva following one shocked child through fluid bolus therapy with reassessment after each aliquot, the transition to vasoactive support for fluid-refractory shock, cold versus warm shock phenotyping, the FEAST caution, the dopamine downgrade, and early retrieval in a resource-limited setting.
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Target exams
Stage 1 — The first bolus
The child weighs 18 kilograms. The heart rate is 165 per minute, capillary refill is 4 seconds, skin is cool and mottled, pulses are weak, blood pressure is 75/45 mmHg, and the child is drowsy but responsive. [4]
Examiner: "What is your immediate fluid management and why?" [2]
I confirm septic shock from the integrated assessment: tachycardia, prolonged capillary refill, weak pulses, mottled skin, hypotension and altered consciousness. I give 10 to 20 mL per kilogram of isotonic crystalloid — 0.9 per cent sodium chloride or Hartmann solution — over 5 to 20 minutes. For this 18 kilogram child, that is 180 to 360 mL per aliquot. Before giving it, I state the improvement I expect: heart rate falling toward normal, shorter capillary refill, stronger pulses, warmer skin and improved interaction. I do not give it as a slow gravity drip — I use a push-pull technique or rapid infuser. [2] [3]
Examiner probes and model responses — Stage 1ShowHide
Probe: "Why not give all the fluid at once?" Because the first-hour ceiling of 40 to 60 mL per kilogram is a maximum to reach through reassessed aliquots, not a single push. The purpose of the aliquot is to give, reassess, and decide. Giving all fluid at once removes the decision point and risks driving the child onto the flat portion of the Starling curve without detecting the signs of overload. [2] [9]
Probe: "What fluid do you choose?" Isotonic crystalloid — either 0.9 per cent sodium chloride or a balanced solution such as Hartmann or Plasma-Lyte. I avoid hypotonic fluids because they do not stay intravascular and can cause hyponatraemia. Albumin is not routinely first-line for the initial bolus. [2] [3]
References5ShowHide
- [1]Maitland K, Kiguli S, Opoka RO, et al. Mortality after fluid bolus in African children with severe infection The New England journal of medicine, 2011.PMID 21615299
- [2]Weiss, Scott L Surviving Sepsis Campaign International Guidelines for the Management of Sepsis and Septic Shock in Children 2026 Pediatric critical care medicine : a journal of the Society of Critical Care Medicine and the World Federation of Pediatric Intensive and Critical Care Societies, 2026.PMID 41869844
- [3]Weiss, Scott L Surviving Sepsis Campaign International Guidelines for the Management of Septic Shock and Sepsis-Associated Organ Dysfunction in Children Pediatric critical care medicine : a journal of the Society of Critical Care Medicine and the World Federation of Pediatric Intensive and Critical Care Societies, 2020.PMID 32032273
- [4]Bjorklund A, Resch J, Slusher T Pediatric Shock Review Pediatrics in review, 2023.PMID 37777656
- [9]Gupta S, Sankar J Advances in Shock Management and Fluid Resuscitation in Children Indian journal of pediatrics, 2023.PMID 36715864