GP KFPs / SAQs · musculoskeletal-presentations
Low back pain — KFP-style written assessment
KFP-style staged scenarios on low back pain: diagnostic triage and first-line care for acute nonspecific pain, the drug evidence stripped of habit (PACE, Machado, Goldberg, OPAL), sciatica and the surgery-timing decision, and recognising cauda equina syndrome fast enough to matter.
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RACGP KFPRACGP AKTMRCGP AKT
Prompt
Low back pain: triage, treat what works, and never miss the emergency
KFP 1 (10 marks)
A 44-year-old warehouse worker presents on day three of severe low back pain after shifting pallets. He has no leg pain, no neurological symptoms, no fever, no cancer history, no steroid use, and no significant trauma beyond the lift. He wants an MRI "to see what's torn" and a sick certificate for four weeks off work. [1]
- State your diagnostic triage for this presentation with approximate proportions, naming what must be actively excluded. (3) [1]
- He asks why you will not order an MRI. Give the trial-based justification. (3) [4]
- Outline first-line management, including one trial result that supports each element. (4) [2][3]
Model answers
- Triage allocates presentations to nonspecific low back pain (90–95% of cases), radicular syndrome (about 5–10%), or specific spinal pathology (below 1% — fracture, malignancy, infection, axial spondyloarthritis, cauda equina). His presentation — mechanical pattern, no deficit, no systemic features — sits in the nonspecific category after excluding non-spinal causes; back pain is a symptom, not a diagnosis. [1]
- A systematic review of six randomised trials (n = 1804) found no significant difference in pain or function between immediate lumbar imaging and usual care without immediate imaging at short- or long-term follow-up; clinicians should refrain from routine immediate lumbar imaging without features suggesting serious underlying condition. Imaging also risks cascades: rapid-MRI strategies produced identical outcomes but more spine operations than radiography. [4]
- Advice to stay active (small but real gains in pain relief and function versus bed rest), heat wrap (moderate-quality first-line nonpharmacologic therapy per ACP), and an NSAID at lowest effective dose if needed — NSAIDs beat placebo across 35 trials, though with clinically unimportant average effect (NNT 6) and 2.5-fold more gastrointestinal reactions; paracetamol is not indicated (PACE: median recovery 17 vs 17 vs 16 days versus placebo, HR 0.99). Encourage early return to modified duties. [2][3]
References8ShowHide
- [1]Bardin LD et al. Diagnostic triage for low back pain: a practical approach for primary care. The Medical journal of Australia, 2017.PMID 28359011
- [2]Williams CM et al. Efficacy of paracetamol for acute low-back pain: a double-blind, randomised controlled trial. Lancet (London, England), 2014.PMID 25064594
- [3]Machado GC et al. Non-steroidal anti-inflammatory drugs for spinal pain: a systematic review and meta-analysis. Annals of the rheumatic diseases, 2017.PMID 28153830
- [4]Chou R et al. Imaging strategies for low-back pain: systematic review and meta-analysis. Lancet (London, England), 2009.PMID 19200918
- [5]Chiu CC et al. The probability of spontaneous regression of lumbar herniated disc: a systematic review. Clinical rehabilitation, 2015.PMID 25009200
- [6]Peul WC et al. Surgery versus prolonged conservative treatment for sciatica. The New England journal of medicine, 2007.PMID 17538084
- [7]Conte A et al. Cauda Equina Syndrome-A 2025 Narrative Review. World neurosurgery, 2026.PMID 42053010
- [8]Najjar E et al. Reassessing the clock in cauda equina syndrome: a systematic review and meta-analysis of surgical timing and outcomes. Neurosurgical review, 2026.PMID 41991105