Anaes · Paediatric anaesthesia
Paediatric airway anatomy, equipment sizing, and ETT selection
Also known as Paediatric airway · Paediatric ETT sizing · Cuffed versus uncuffed endotracheal tube · Paediatric laryngoscopy · Difficult paediatric airway
The paediatric airway differs from the adult airway in ways that change every step of airway management: the larynx is high and anterior, the tongue and occiput are large, the epiglottis is long and floppy, and the cricoid is classically the narrowest point. Endotracheal tube selection follows the Cole formulae (uncuffed and cuffed), with depth approximated as three times the internal diameter. Modern cuffed tubes are safe and reduce tube exchange. The defining emergency is laryngospasm progressing to bradycardia and arrest. This topic covers anatomy, equipment sizing, cuffed versus uncuffed evidence, positioning, the difficult and syndromic airway, and laryngospasm management for the ANZCA Final Examination and its cross-exam equivalents.
Practise this topic
On this page
Study tools
Your progress
Saved on this device.
Target exams
Red flags
- The infant desaturates rapidly after apnoea: a reduced functional residual capacity and an oxygen consumption around twice the adult value (approximately 6 mL/kg/min) make the safe apnoea time short. Preoxygenation is essential.
- The paediatric larynx is high and anterior (infant C3 to C4, adult C5 to C6), the tongue is large relative to the oral cavity, and the epiglottis is long, floppy and U-shaped. A straight (Miller) blade and a different laryngoscopy technique are often required.
- The cricoid cartilage is classically the narrowest point of the paediatric airway (not the vocal cords), giving the airway a funnel shape; an uncuffed tube that passes the cords can still obstruct at the cricoid.
- Laryngospasm is the canonical cause of paediatric perioperative cardiac arrest from airway events: it escalates from partial obstruction to complete closure, bradycardia, and arrest within minutes if unrecognised.
- A concurrent upper respiratory tract infection multiplies the risk of laryngospasm (odds ratio about 2), and an airway anomaly multiplies it further (odds ratio about 3.4).
Meet the patient
A previously well two-year-old develops stridor, then silent obstruction, during a sevoflurane induction for myringotomy. The saturation is falling fast. The inhalational circuit is on, the parents are watching, and you have seconds before the heart rate drops with it.[1]
This is laryngospasm, the canonical paediatric airway crisis, and it escalates from partial closure to complete closure to bradycardia to arrest in minutes. The two facts that govern your next thirty seconds are the two that govern every paediatric airway: the child desaturates in seconds, and bradycardia is hypoxia until proven otherwise. Hold those, and the anatomy, the formulae, and the ladder below all slot into place.[1]
References6ShowHide
- [1]Shi F, Xiao Y, Xiong W, Zhou Q, Huang X Cuffed versus uncuffed endotracheal tubes in children: a meta-analysis J Anesth, 2016.PMID 26296534
- [2]Flick RP, Wilder RT, Pieper SF, et al. Risk factors for laryngospasm in children during general anesthesia Paediatr Anaesth, 2008.PMID 18315633
- [3]Mihara T, Uchimoto K, Morita S, Goto T The efficacy of lidocaine to prevent laryngospasm in children: a systematic review and meta-analysis Anaesthesia, 2014.PMID 24992191
- [4]Sikich N, Lerman J Development and psychometric evaluation of the pediatric anesthesia emergence delirium scale Anesthesiology, 2004.PMID 15114210
- [5]Davidson AJ, Disma N, de Graaff JC, et al. Neurodevelopmental outcome at 2 years of age after general anaesthesia and awake-regional anaesthesia in infancy (GAS): an international multicentre, randomised controlled trial Lancet, 2016.PMID 26507180
- [6]McCann ME, de Graaff JC, Dorris L, et al. Neurodevelopmental outcome at 5 years of age after general anaesthesia or awake-regional anaesthesia in infancy (GAS): an international, multicentre, randomised, controlled equivalence trial Lancet, 2019.PMID 30782342