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Anaes TopicsVascular anaesthesia

Anaes · Vascular anaesthesia

AAA open repair versus EVAR: anaesthetic decision matrix

Also known as AAA open repair versus EVAR: anaesthetic decision matrix

Exam-exhaustive open AAA vs EVAR anaesthesia for ANZCA Final.

high5 referencesUpdated 26 July 20269 min readVerification in progress

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ANZCAFRCAABAEDAICFCAI

Red flags

  • Open AAA is major haemodynamic stress with clamp physiology.
  • EVAR is less invasive but still high cardiac-risk population.
  • Ruptured AAA: permissive hypotension until control.
  • RCRI stratifies perioperative cardiac risk.
  • Declamping shock is the classic pivot.
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Practise this topic10 MCQs with explanations

Target exams

ANZCAFRCAABAEDAICFCAI

Red flags

  • Open AAA is major haemodynamic stress with clamp physiology.
  • EVAR is less invasive but still high cardiac-risk population.
  • Ruptured AAA: permissive hypotension until control.
  • RCRI stratifies perioperative cardiac risk.
  • Declamping shock is the classic pivot.
The one-line answer

Open AAA repair is major haemodynamic stress built around aortic cross-clamp and declamp physiology; EVAR is less invasive but still a high-risk vascular patient. Ruptured AAA uses permissive hypotension until aortic control. Lead with the one-line priority, then the exact numbers, then the crisis pivot — "clamp up, afterload up; clamp down, pressure down."[3][3][5]

Meet the patient

A 72-year-old smoker with a 6 cm infrarenal AAA and a 30-pack-year history is booked for elective open repair. He had a myocardial infarction two years ago. The surgeon plans a midline laparotomy with an infrarenal clamp. Before induction you confirm the arterial line, the large-bore access, the blood in theatre, and the cell salvage — because moment clamp goes on, this becomes a cardiac and vascular physiology problem, not a hernia.[1]

Two questions frame every AAA list: can the heart stand the clamp and the unclamp? and is this elective open, elective EVAR, or a rupture? The answer decides the monitoring, the access, and the disposition.[1]

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References5ShowHide
  1. [1]Lee TH, Marcantonio ER, Mangione CM, et al. Derivation and prospective validation of a simple index for prediction of cardiac risk of major noncardiac surgery. Circulation, 1999.PMID 10477528
  2. [2]Devereaux PJ, Yang H, Yusuf S, et al. Effects of extended-release metoprolol succinate in patients undergoing non-cardiac surgery (POISE trial): a randomised controlled trial. Lancet, 2008.PMID 18479744
  3. [3]EVAR trial participants Endovascular aneurysm repair versus open repair in patients with abdominal aortic aneurysm (EVAR trial 1): randomised controlled trial. Lancet, 2005.PMID 15978925
  4. [4]Patel R, Sweeting MJ, Powell JT, Greenhalgh RM Endovascular versus open repair of abdominal aortic aneurysm in 15-years' follow-up of the UK endovascular aneurysm repair trial 1 (EVAR trial 1): a randomised controlled trial. Lancet, 2016.PMID 27743617
  5. [5]Loggi S, Mininno N, Damiani E, et al. Changes in the sublingual microcirculation following aortic surgery under balanced or total intravenous anaesthesia: a prospective observational study. BMC Anesthesiol, 2019.PMID 30611197

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NextAortic cross-clamp physiology: clamp-on and clamp-offVascular anaesthesia