Anaes · Applied cardiovascular & respiratory physiology
Microcirculation & control of blood flow
Also known as Microcirculation · Capillary exchange · Starling forces · Autoregulation · Local blood flow control · Hypoxic pulmonary vasoconstriction
The microcirculation — the arterioles, capillaries and venules — is where blood flow is actually distributed and where exchange with the tissues occurs. The framework rests on five exam-critical ideas: local blood flow is controlled chiefly at the arteriolar and precapillary sphincter level, by metabolic feedback (CO2, hydrogen ion, potassium, adenosine, lactate) that matches flow to demand, by myogenic tone, and by endothelial factors (nitric oxide, prostaglandins, endothelin); capillary exchange is governed by the Starling forces — the balance of hydrostatic and oncotic pressures across the capillary wall — with filtration at the arterial end and reabsorption at the venous end; oedema results from a disturbance of one of four factors (raised hydrostatic pressure, low plasma oncotic pressure, raised capillary permeability, lymphatic obstruction); the lung is the exception that proves metabolic control, constricting rather than dilating in response to hypoxia (hypoxic pulmonary vasoconstriction); and microcirculatory dysfunction — a leaky, leaky-flow state with shunting — explains why a normal macroscopic blood pressure does not guarantee tissue oxygenation in sepsis. Built on the microvascular-failure-in-septic-shock review (Popovich 2026), the carbon-dioxide-vasodilator review (Duse 2026), the transvascular-exchange/revised-Starling review (Alamilla-Sanchez 2023), the capillary-hydration study (Pstras 2022), the perioperative-renal-microcirculation review (Li 2026), and the pulmonary-vascular-tone study (Maier 2026).
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- Local blood flow is controlled by metabolic feedback at the arteriole and precapillary sphincter — active metabolites such as CO2, hydrogen ion, potassium, adenosine and lactate dilate the arteriole, matching flow to demand. This is why hypercarbia and tissue underperfusion cause vasodilation.
- Starling filtration is driven by capillary hydrostatic pressure and opposed by plasma oncotic (colloid) pressure; net filtration occurs at the arterial end and reabsorption at the venous end, with the lymphatics returning the small net filtrate.
- Oedema has four mechanisms — raised capillary hydrostatic pressure (heart failure, venous obstruction), low plasma oncotic pressure (hypoalbuminaemia), raised capillary permeability (sepsis, allergy, inflammation), and lymphatic obstruction — each with different fluid management implications.
- Hypoxic pulmonary vasoconstriction is the exception to metabolic vasodilation: pulmonary arterioles constrict in response to alveolar hypoxia, redirecting flow to better-ventilated lung. Volatile anaesthetics, vasodilators and low SVR all impair it, worsening one-lung ventilation hypoxaemia.
- A normal systemic blood pressure does not guarantee tissue perfusion: in septic and vasodilatory shock the microcirculation leaks and shunts, so flow bypasses exchange capillaries and oxygen extraction fails despite an adequate macroscopic pressure.
Meet the patient
A septic patient in the ICU has a mean arterial pressure of 75, maintained with noradrenaline. The lactate is rising, the urine output is falling, and the skin is mottled despite a pressure that looks adequate on the monitor.[1]
This is microcirculatory failure hiding behind a normal macroscopic blood pressure. The endothelium is leaking, the glycocalyx is shredded, and capillaries are shunting blood past unperfused tissue. The cuff pressure is necessary but not sufficient — the microcirculation decides whether the cells get oxygen.[1]
References6ShowHide
- [1]Popovich I, et al. On the inevitability of microvascular failure in septic shock and other vasodilatory conditions Crit Care, 2026.PMID 42343425
- [2]Duse DA. Carbon dioxide is a triple vasodilator Cardiovasc Res, 2026.PMID 42334380
- [3]Alamilla-Sanchez ME, et al. Advances in the Physiology of Transvascular Exchange and A New Look At Rational Fluid Prescription Int J Gen Med, 2023.PMID 37408844
- [4]Pstras L, et al. Vascular refilling coefficient is not a good marker of whole-body capillary hydraulic conductivity in hemodialysis patients: insights from a simulation study Sci Rep, 2022.PMID 36088359
- [5]Li S, et al. Pathological triad of perioperative acute kidney injury: renal microcirculatory hypoxia, mitochondrial damage, and immuno-metabolic reprogramming Front Immunol, 2026.PMID 42338604
- [6]Maier LE, et al. The influence of metaboreflex activation on pulmonary pressure with combined chemoreflex activation in acute and chronic hypoxia J Physiol, 2026.PMID 42306962