Small Bowel Obstruction (SBO)
Summary
Small Bowel Obstruction (SBO) is a mechanical blockage of the small intestine preventing the normal passage of intestinal contents. It is a common surgical emergency, with adhesions from previous abdominal surgery being the most common cause (~60%), followed by hernias (~20%). Clinical features include colicky abdominal pain, vomiting, abdominal distension, and constipation. Diagnosis is primarily clinical and radiological (Abdominal X-ray shows dilated small bowel loops). CT Abdomen is the gold standard for assessing cause and identifying complications. Management involves resuscitation ("Drip and Suck"), with conservative management (NG tube, IV fluids, Gastrografin challenge) often successful for adhesive SBO. Surgery is indicated for signs of strangulation, closed loop obstruction, hernias, failure of conservative management, or complete obstruction. [1,2]
Clinical Pearls
"Never Let the Sun Set on a Bowel Obstruction": This old adage reminds clinicians that delayed treatment of SBO, especially with strangulation, leads to bowel necrosis and significantly increased mortality.
Adhesions = Most Common Cause: 60% of SBO is due to adhesions from previous surgery. No hernia? Ask about abdominal operations.
Vomiting is Early, Constipation is Late: Unlike large bowel obstruction. In proximal SBO, vomiting is profuse and early. Distension may be minor.
Gastrografin Challenge: Diagnostic and potentially therapeutic. Passage of contrast to colon within 24 hours predicts resolution; can accelerate resolution.
Incidence
- Common Surgical Emergency: ~15-20% of emergency laparotomies.
- Age: All ages. Risk increases with previous surgery.
Causes
| Cause | Prevalence | Notes |
|---|---|---|
| Adhesions | 60% | Previous abdominal/pelvic surgery. May occur years later. |
| Hernias (Incarcerated) | 20% | Inguinal, Femoral, Incisional, Umbilical, Internal. Examine all hernia orifices! |
| Malignancy | 5-10% | Primary small bowel tumour (rare), Metastatic (peritoneal carcinomatosis). |
| Crohn's Disease | 5% | Stricture from chronic inflammation. |
| Volvulus | less than 5% | Rare in small bowel (more common in colon/sigmoid). |
| Gallstone Ileus | less than 5% | Large gallstone erodes into duodenum, obstructs at ileocaecal valve. Rigler's Triad (Pneumobilia, SBO, Ectopic gallstone). |
| Intussusception / Foreign Body / Stricture | less than 5% | Various other causes. |
Mechanism
- Mechanical Obstruction: Lumen of small bowel is blocked by adhesive band, hernia, tumour, etc.
- Proximal Bowel Dilation: Gas and fluid accumulate proximal to obstruction.
- Secretion Accumulation: ~8-10L of fluid secreted into GI tract daily cannot pass → Fluid sequestration.
- Dehydration & Electrolyte Loss: Vomiting + Third-space losses → Hypovolaemia, Hypochloraemia, Hypokalaemia, Metabolic Alkalosis (if prolonged vomiting).
- Bowel Wall Oedema: Venous congestion → Wall swelling → Worsens obstruction.
- Strangulation (If Blood Supply Compromised):
- Venous obstruction first → Congestion, Haemorrhagic infarction.
- Arterial obstruction → Ischaemia → Necrosis → Perforation → Peritonitis.
- Bacterial Translocation: Gut barrier breakdown → Sepsis.
Types
| Type | Description |
|---|---|
| Simple | Single point of obstruction. Proximal dilation. |
| Closed Loop | Two points of obstruction (e.g., adhesive band traps a loop). High risk of strangulation. Rapidly progresses. |
| Complete | No passage of gas or liquid distally. |
| Partial (Incomplete) | Some passage. May pass flatus. |
| Strangulated | Blood supply compromised. Surgical emergency. |
| Condition | Key Features |
|---|---|
| Small Bowel Obstruction | Colicky pain, Vomiting, Distension, History of surgery/hernia. Central dilated loops on AXR. |
| Large Bowel Obstruction | Distension (massive), Constipation (early), Vomiting (late). Peripheral dilated loops. May have caecal dilatation. |
| Paralytic Ileus | Post-operative. Diffuse dilation of small and large bowel. No mechanical cause. Bowel sounds absent. |
| Pseudo-Obstruction (Ogilvie's Syndrome) | Large bowel dilatation without mechanical cause. Often elderly, hospitalised. |
| Gastroenteritis | Vomiting, Diarrhoea (not constipation). No distension. No dilated loops. |
| Mesenteric Ischaemia | Severe pain out of proportion to examination. AF, Lactate elevation. May have SBO features later. |
Symptoms (Classic Triad: Pain, Vomiting, Constipation)
| Symptom | Notes |
|---|---|
| Colicky Abdominal Pain | Central. Comes in waves. Associated with visible peristalsis in thin patients. |
| Vomiting | Early and profuse in proximal SBO ("Coffee ground" or bilious). Less prominent in distal SBO. |
| Absolute Constipation | No passage of faeces or flatus. Late sign. Early in distal SBO. |
| Abdominal Distension | More prominent in distal SBO. Minimal in proximal obstruction. |
Signs
| Sign | Notes |
|---|---|
| Distension | Generalised abdominal distension. Tympanic to percussion. |
| Bowel Sounds | High-pitched, tinkling ("obstructed") early. May become silent late (ileus or ischaemia). |
| Visible Peristalsis | In thin patients – "Ladder pattern". |
| Tenderness | Generalised mild tenderness OK. Localised tenderness / Peritonism = Concern for strangulation. |
| Hernias | Always examine hernia orifices (Groins, Umbilicus, Scars). |
| Shock | Tachycardia, Hypotension (late – indicates severe dehydration or strangulation). |
Blood Tests
| Test | Findings |
|---|---|
| FBC | WCC elevated (Leucocytosis – infection/strangulation). Hb raised (Haemoconcentration). |
| U&E | Raised Urea/Creatinine (Dehydration). Hypokalaemia (Vomiting). Hypochloraemia. |
| Lactate | Elevated = Bowel Ischaemia. Urgent surgical indicator. |
| ABG | Metabolic Alkalosis (Vomiting) or Metabolic Acidosis (if ischaemia/shock). |
| Amylase | May be mildly elevated. Rule out Pancreatitis. |
| Group & Save | In case of surgery. |
Imaging
| Imaging | Findings |
|---|---|
| Abdominal X-ray (AXR) | Central dilated small bowel loops (>3cm). Valvulae conniventes (lines cross entire lumen). Multiple air-fluid levels on erect film. Collapsed large bowel distally. |
| CT Abdomen with Contrast (Gold Standard) | Identifies cause (adhesive band, hernia, tumour). Transition Point (site of obstruction). Signs of ischaemia (Thickened bowel wall, Mesenteric haziness, Reduced enhancement, Pneumatosis, Portal venous gas). Closed loop. |
| Gastrografin (Water-Soluble Contrast) | Given orally or via NG. Passage to colon in 4-24h = Likely resolution. Can be therapeutic (osmotic effect). |
Rigler's Triad (Gallstone Ileus)
- Pneumobilia (Air in biliary tree).
- Small Bowel Obstruction.
- Ectopic gallstone (usually at Ileocaecal valve).
Management Algorithm
SMALL BOWEL OBSTRUCTION
(Colicky Pain, Vomiting, Distension)
↓
RESUSCITATION ("DRIP AND SUCK")
- IV Access, Fluids (Normal Saline / Hartmann's)
- NG Tube (Large Bore, Free Drainage – Decompression)
- Urinary Catheter (Monitor Output)
- NBM
- Analgesia (IV Morphine)
- Correct Electrolytes (K+)
↓
ASSESS FOR STRANGULATION / EMERGENCY SURGERY INDICATIONS:
┌───────────────────────────────────────────────────────┐
│ INDICATORS FOR URGENT SURGERY: │
│ - Peritonitis (Guarding, Rebound, Rigidity) │
│ - Incarcerated / Irreducible Hernia │
│ - Shock (Tachycardia, Hypotension) │
│ - Elevated Lactate │
│ - CT: Closed Loop, Ischaemia Signs (Pneumatosis, etc) │
│ - Failure of Conservative Management (48-72h) │
│ - Complete Obstruction │
└───────────────────────────────────────────────────────┘
┌────────────────┴────────────────┐
YES NO
↓ ↓
EMERGENCY SURGERY CONSERVATIVE MANAGEMENT
(Laparotomy / Laparoscopy) (Adhesive SBO, Partial)
- Adhesiolysis ↓
- Resection if ischaemic GASTROGRAFIN CHALLENGE
- Hernia Repair (100ml Gastrografin via NG)
↓
X-RAY at 4-8h and 24h:
Contrast in Colon?
┌───────────┴───────────┐
YES NO
↓ ↓
LIKELY TO LIKELY TO NEED
RESOLVE SURGERY
→ Continue Conservative
→ Diet as tolerated → Proceed to OR
Conservative Management ("Drip and Suck")
| Component | Details |
|---|---|
| IV Fluids | Crystalloid (Hartmann's / Normal Saline). Replace losses. Aim UOP >0.5ml/kg/hr. |
| NG Tube | Large bore. Free drainage. Decompresses stomach. Reduces vomiting and aspiration risk. |
| NBM | Nil by mouth until obstruction resolved. |
| Electrolyte Correction | Especially Potassium. |
| Monitoring | Regular observations. Abdominal examination. Repeat bloods (Lactate). |
| Gastrografin Challenge | Diagnostic and therapeutic. Osmotic draw of fluid into lumen may help resolve partial obstruction. Passage to colon = Good prognosis for resolution. |
Surgical Management
| Indication | Procedure |
|---|---|
| Strangulation / Peritonitis | Emergency Laparotomy. Resect necrotic bowel. Anastomosis or Stoma. |
| Incarcerated Hernia | Hernia repair +/- Bowel resection if ischaemic. |
| Adhesive SBO (Failure of Conservative) | Laparotomy or Laparoscopic Adhesiolysis. |
| Malignancy | Resection / Bypass / Stenting (Palliative). |
| Gallstone Ileus | Enterotomy, Stone removal. Consider Cholecystectomy later. |
| Complication | Notes |
|---|---|
| Strangulation / Bowel Necrosis | Requires resection. High mortality if delayed. |
| Perforation | Peritonitis. Sepsis. Emergency surgery. |
| Aspiration Pneumonia | Due to vomiting. NG decompression reduces risk. |
| Electrolyte Disturbance | Hypokalaemia, Metabolic Alkalosis. |
| Dehydration / AKI | From third-space losses and vomiting. |
| Short Bowel Syndrome | If extensive resection required. |
| Recurrence | Adhesions can recur after adhesiolysis. |
- Adhesive SBO: ~70-80% resolve with conservative management.
- Mortality: Low for simple obstruction (less than 5%). Higher for strangulated/perforated bowel (15-25%).
- Recurrence: ~20-30% for adhesive SBO.
Key Guidelines
| Guideline | Organisation | Key Recommendations |
|---|---|---|
| Bologna Guidelines (WSES) | World Society of Emergency Surgery | CT for diagnosis. Conservative for adhesive SBO. Gastrografin. Indications for surgery. |
Landmark Trials
- Gastrografin Studies: Demonstrated faster resolution of partial adhesive SBO with water-soluble contrast.
What is Small Bowel Obstruction?
It is a blockage in the small intestine (the long, thin tube that carries food from the stomach to the large bowel). Food and liquid cannot pass through, causing pain, vomiting, and swelling of the tummy.
What causes it?
The most common cause is scar tissue (adhesions) from previous surgery. Hernias are the second most common cause.
How is it treated?
We put you on a drip for fluids and place a tube through your nose into your stomach to drain the backed-up fluid. This lets the bowel rest and often allows the blockage to settle. Sometimes a special dye drink (Gastrografin) can help the bowel unblock.
Will I need surgery?
Not always. Many blockages resolve with rest and fluids. But if the blockage doesn't clear, or if the blood supply to the bowel is at risk (strangulation), you will need an operation to fix it.
Primary Sources
- Ten Broek RPG, et al. Bologna guidelines for diagnosis and management of adhesive small bowel obstruction (ASBO). World J Emerg Surg. 2018;13:24. PMID: 29946347.
- Drago A, Rosenthal MJ. Small bowel obstruction. Clin Geriatr Med. 2017;33(1):145-158.
Common Exam Questions
- Most Common Cause: "Most common cause of SBO in adults?"
- Answer: Adhesions (from previous surgery).
- X-ray Findings: "Describe AXR findings in SBO."
- Answer: Centrally located dilated small bowel loops (>3cm). Valvulae conniventes (lines across full width of bowel). Air-fluid levels (on erect film).
- Strangulation Signs: "Signs of strangulation on CT?"
- Answer: Thickened bowel wall, Mesenteric haziness, Reduced bowel wall enhancement, Pneumatosis intestinalis, Portal venous gas.
- Gastrografin: "Role of Gastrografin in adhesive SBO?"
- Answer: Diagnostic (if reaches colon = likely resolution) and potentially therapeutic (osmotic effect).
Viva Points
- Bologna Guidelines: Know indications for surgery vs conservative management.
- Closed Loop Obstruction: Explain high risk of ischaemia and need for urgent surgery.
Medical Disclaimer: MedVellum content is for educational purposes and clinical reference. Clinical decisions should account for individual patient circumstances. Always consult appropriate specialists.