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EMERGENCY

Major Depressive Disorder

High EvidenceUpdated: 2025-12-22

On This Page

Red Flags

  • Active suicidal ideation with plans/intent
  • Psychotic features (delusions, hallucinations)
  • Severe self-neglect
  • Catatonia
  • Recent suicide attempt
Overview

Major Depressive Disorder

1. Topic Overview

Summary

Major Depressive Disorder (MDD) is a common, serious mood disorder characterised by persistent low mood, anhedonia (loss of pleasure), and associated cognitive, behavioural, and physical symptoms. Affecting approximately 5% of the adult population globally, depression is a leading cause of disability worldwide. The diagnosis requires symptoms to be present for at least two weeks and to cause significant functional impairment. Treatment involves a biopsychosocial approach: antidepressants (SSRIs first-line), psychological therapy (CBT, IPT), and addressing social factors. Early recognition and treatment significantly improves outcomes.

Key Facts

  • Prevalence: 5-7% lifetime prevalence in adults
  • Sex Ratio: 2:1 female to male
  • Peak Onset: 20s-30s, but can occur at any age
  • Recurrence: 50% after first episode, 80% after two episodes
  • Disability: Leading cause of disability worldwide (WHO)
  • Mortality: 15-fold increased suicide risk; increased cardiovascular mortality
  • Treatment Response: 60-70% respond to first antidepressant

Clinical Pearls

High-Yield Points:

  • Always assess suicide risk directly and thoroughly
  • Core symptoms: Low mood, Anhedonia, Anergia (lack of energy)
  • Biological symptoms suggest severity: early morning wakening, diurnal variation, psychomotor changes
  • SSRIs take 2-4 weeks for effect; warn patients about this delay
  • Combination of medication + therapy is more effective than either alone
  • Rule out organic causes: hypothyroidism, anaemia, medication-induced

Why This Matters Clinically

Depression is one of the most common conditions encountered in primary care and all medical specialties. Untreated depression increases mortality from suicide and comorbid medical conditions. Depression worsens outcomes in cardiovascular disease, diabetes, and cancer. Early recognition and evidence-based treatment can be life-saving.


2. Epidemiology

Prevalence and Incidence

MetricValue
Lifetime Prevalence15-20%
Point Prevalence5-7%
Incidence3% per year
Gender RatioFemale:Male 2:1

Risk Factors

Non-Modifiable:

  • Female sex
  • Family history of depression (2-3x increased risk)
  • Personal history of depression
  • Adverse childhood experiences

Modifiable:

  • Social isolation and loneliness
  • Unemployment
  • Chronic medical illness
  • Substance misuse (alcohol, cannabis)
  • Sleep disturbance

Precipitating Factors:

  • Major life events (bereavement, relationship breakdown)
  • Physical illness diagnosis
  • Postpartum period
  • Medication (beta-blockers, steroids, isotretinoin)

3. Pathophysiology

Biological Models

Monoamine Hypothesis:

  • Reduced serotonin, norepinephrine, and dopamine neurotransmission
  • Basis for SSRI, SNRI, and TCA mechanisms
  • Oversimplified but clinically useful model

Neuroplasticity Model:

  • Reduced BDNF (brain-derived neurotrophic factor)
  • Hippocampal volume reduction
  • Impaired neurogenesis

HPA Axis Dysregulation:

  • Chronic stress leads to cortisol elevation
  • Hippocampal damage and feedback loop disruption

Inflammation:

  • Elevated inflammatory markers (IL-6, CRP)
  • Bidirectional relationship with physical illness

Psychological Models

  • Cognitive Model (Beck): Negative automatic thoughts, cognitive distortions
  • Learned Helplessness (Seligman): Belief in inability to control outcomes
  • Behavioural Model: Reduced positive reinforcement from environment

4. Clinical Presentation

Diagnostic Criteria (ICD-11 / DSM-5)

Core Symptoms (at least one required):

  1. Depressed mood (most of the day, nearly every day)
  2. Anhedonia (loss of interest or pleasure)

Additional Symptoms (5 or more total including core): 3. Weight/appetite change (increase or decrease) 4. Sleep disturbance (insomnia or hypersomnia) 5. Psychomotor agitation or retardation 6. Fatigue or loss of energy 7. Feelings of worthlessness or excessive guilt 8. Concentration difficulties, indecisiveness 9. Recurrent thoughts of death or suicide

Duration: ≥2 weeks

Impact: Causes significant distress or functional impairment

Biological Symptoms (Indicate Severity)

Severity Classification

SeveritySymptom CountFunction
Mild5 symptomsMinor impairment
Moderate6-7 symptomsModerate impairment
Severe8-9 symptomsMarked impairment, may have psychotic features

Psychotic Depression


Early Morning Wakening
Waking 2-3 hours early, unable to return to sleep
Diurnal Variation
Mood worst in morning, improves through day
Psychomotor Changes
Slowed movements (retardation) or agitation
Weight Loss
Significant, unintentional
Loss of Libido
Common presentation.
5. Clinical Examination

Mental State Examination

DomainFindings in Depression
AppearanceUnkempt, poor self-care, reduced eye contact
BehaviourPsychomotor retardation or agitation
SpeechReduced rate and volume, increased latency
Mood"Low", "hopeless", "empty" (subjective)
AffectFlattened, restricted, tearful
Thought ContentGuilt, worthlessness, hopelessness, suicidal ideation
CognitionPoor concentration ("pseudodementia" in severe cases)
InsightUsually preserved

Risk Assessment (ESSENTIAL)

Suicide Risk Factors:

  • Previous suicide attempt (strongest predictor)
  • Current suicidal ideation with plans/intent
  • Access to means
  • Male sex, older age
  • Social isolation
  • Recent loss
  • Comorbid substance misuse
  • Hopelessness

Protective Factors:

  • Social support
  • Dependent children
  • Religious beliefs
  • Therapeutic alliance

6. Investigations

Purpose: Rule Out Organic Causes

InvestigationRationale
FBCAnaemia, infection
TFTsHypothyroidism
LFTsAlcohol misuse
U&Es, GlucoseMetabolic causes
CalciumHypercalcaemia
B12, FolateDeficiency
Drug ScreenSubstance-induced

Rating Scales (Severity Monitoring)

  • PHQ-9: 9 questions, scores 0-27 (Mild 5-9, Moderate 10-14, Severe ≥15)
  • HAMD: Hamilton Depression Rating Scale (clinician-rated)
  • BDI: Beck Depression Inventory

7. Management

NICE Stepped Care Model

Step 1 (Sub-threshold/Mild):

  • Assessment and active monitoring
  • Self-help resources, exercise
  • Sleep hygiene

Step 2 (Mild-Moderate):

  • Low-intensity psychological intervention (guided self-help, computerised CBT)
  • Exercise programmes

Step 3 (Moderate-Severe):

  • High-intensity psychological therapy (CBT, IPT, BA)
  • Antidepressant medication

Step 4 (Severe/Complex/Treatment-Resistant):

  • Specialist mental health services
  • Crisis teams
  • Inpatient care if high risk

Pharmacological Treatment

First-Line: SSRIs

DrugStarting DoseTarget DoseNotes
Sertraline50 mg OD100-200 mgNICE first choice, safe in cardiac disease
Fluoxetine20 mg OD20-60 mgLong half-life, activating
Citalopram10-20 mg OD20-40 mgQTc prolongation risk, max 20mg if >5
Escitalopram5-10 mg OD10-20 mgPotent, well tolerated

Second-Line/Alternatives:

ClassExamplesWhen to Use
SNRIVenlafaxine, DuloxetineSSRI failure, pain comorbidity
Mirtazapine15-45 mgInsomnia, weight loss, nausea with SSRIs
TCAAmitriptylineSpecialist use, chronic pain

Treatment Principles:

  • Start low, go slow (50% dose initially)
  • Allow 2-4 weeks for effect
  • Continue 6-9 months after remission (first episode)
  • 2+ years if recurrent depression
  • Taper slowly when stopping (discontinuation syndrome)

Psychological Therapies

TherapyEvidenceFormat
CBTStrong (NICE recommended)12-16 sessions
IPTStrong12-16 sessions
Behavioural ActivationStrongFocuses on activity scheduling
CounsellingModerateSupportive

Electroconvulsive Therapy (ECT)

Indications:

  • Severe depression with life-threatening features (severe self-neglect, catatonia)
  • Psychotic depression
  • Treatment-resistant depression
  • Rapid response needed

Efficacy: 70-80% response rate in severe depression

Resistant Depression (After 2+ Adequate Trials)

  • Augmentation: Add lithium or aripiprazole
  • Switch antidepressant class
  • Ketamine/esketamine (specialist)
  • ECT
  • TMS (repetitive transcranial magnetic stimulation)

8. Complications

Acute

ComplicationRiskManagement
Suicide15% lifetime in severeRisk assessment, crisis plan
Self-harmCommonSafety planning
Psychosis10-15% of severe casesAntipsychotic + AD

Chronic

  • Recurrent episodes (80% after 2 episodes)
  • Treatment-resistant depression (30%)
  • Functional impairment (work, relationships)
  • Cardiovascular disease (2x risk)
  • Substance misuse (comorbid in 30%)

9. Prognosis

Natural History

  • Episode Duration: 6-12 months untreated
  • With Treatment: Remission in 60-70% within 6 months
  • Recurrence: 50% after first episode

Prognostic Factors

Poor Prognosis:

  • Severe initial episode
  • Psychotic features
  • Chronic duration
  • Comorbid personality disorder
  • Poor social support
  • Substance misuse

Good Prognosis:

  • Acute onset
  • Clear precipitant
  • Good premorbid function
  • Strong social support
  • Treatment adherence

10. Evidence and Guidelines

Key Guidelines

GuidelineOrganisationYear
Depression in AdultsNICE CG902009 (Updated 2022)
APA Practice GuidelineAPA2019
CANMAT GuidelinesCanadian Network2023

Key Evidence

  • STAR*D: Large trial showing ~33% remission with first SSRI, cumulative 67% over 4 steps
  • COMBINE Studies: Psychological + pharmacological = best outcomes

11. Patient/Layperson Explanation

What is Depression?

Depression is more than just feeling sad – it's a medical condition that affects how you think, feel, and function. It involves changes in brain chemistry and can happen to anyone. The key symptoms are feeling persistently low and losing interest in things you used to enjoy.

What causes it?

Depression is caused by a combination of factors:

  • Brain chemistry: Changes in mood-regulating chemicals
  • Genetics: Runs in families
  • Life events: Stress, loss, trauma
  • Physical illness: Chronic conditions can trigger depression

How is it treated?

Treatment works well for most people:

  • Talking therapies: Like CBT, help change unhelpful thought patterns
  • Medication: Antidepressants correct chemical imbalances (not addictive)
  • Lifestyle: Exercise, sleep, social connection all help
  • Most people feel better within 2-3 months with proper treatment

When to seek urgent help

Go to A&E or call 999 if you or someone else:

  • Has thoughts of suicide or self-harm
  • Has made a plan to end their life
  • Feels unable to keep themselves safe

Helplines: Samaritans 116 123 (UK, free, 24/7)


12. References
  1. NICE. Depression in adults: treatment and management (CG90). 2022. nice.org.uk

  2. American Psychiatric Association. Practice Guideline for the Treatment of Patients With Major Depressive Disorder. 2019. psychiatryonline.org

  3. Kennedy SH, et al. Canadian Network for Mood and Anxiety Treatments (CANMAT) 2016 Clinical Guidelines. Can J Psychiatry. 2016;61(9):540-560. PMID: 27486148

  4. Rush AJ, et al. Acute and longer-term outcomes in depressed outpatients requiring one or several treatment steps (STAR*D). Am J Psychiatry. 2006;163(11):1905-1917. PMID: 17074942


Medical Disclaimer: MedVellum content is for educational purposes and clinical reference. It does not replace professional medical judgement. Always verify critical information and consider individual patient factors.

Last updated: 2025-12-22

At a Glance

EvidenceHigh
Last Updated2025-12-22
Emergency Protocol

Red Flags

  • Active suicidal ideation with plans/intent
  • Psychotic features (delusions, hallucinations)
  • Severe self-neglect
  • Catatonia
  • Recent suicide attempt

Clinical Pearls

  • **High-Yield Points:**
  • - Always assess suicide risk directly and thoroughly
  • - Core symptoms: Low mood, Anhedonia, Anergia (lack of energy)
  • - Biological symptoms suggest severity: early morning wakening, diurnal variation, psychomotor changes
  • - SSRIs take 2-4 weeks for effect; warn patients about this delay

Guidelines

  • NICE Guidelines
  • BTS Guidelines
  • RCUK Guidelines