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Gynaecology
General Practice

Endometriosis

High EvidenceUpdated: 2025-12-24

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Red Flags

  • Bowel obstruction
  • Ureteric involvement / Hydronephrosis
  • Severe pain not responding to treatment
  • Ovarian mass (exclude malignancy)
Overview

Endometriosis

1. Clinical Overview

Summary

Endometriosis is a chronic inflammatory condition characterised by the presence of endometrial-like tissue outside the uterine cavity. It affects approximately 10% of reproductive-age women and is a leading cause of chronic pelvic pain, dysmenorrhoea, dyspareunia, and subfertility. Common sites include the ovaries (endometriomas/"chocolate cysts"), pouch of Douglas, uterosacral ligaments, and pelvic peritoneum. Diagnosis is clinical/suspected based on symptoms but confirmed definitively by laparoscopy. Management depends on symptoms and fertility wishes: medical options include hormonal suppression (combined pill, progestogens, GnRH agonists); surgical options include laparoscopic excision or ablation. Endometriosis significantly impacts quality of life and requires multidisciplinary management.

Key Facts

  • Prevalence: ~10% of reproductive-age women; 25-50% of women with infertility
  • Sites: Ovaries (endometriomas); Pouch of Douglas; Uterosacral ligaments; Rectovaginal septum; Bladder; Bowel
  • Symptoms: Chronic pelvic pain; Dysmenorrhoea; Dyspareunia; Subfertility; Cyclical symptoms
  • Diagnosis: Clinical suspicion; Confirmed by laparoscopy + histology
  • Staging: rASRM (revised American Society for Reproductive Medicine) I-IV
  • Treatment: Analgesia; Hormonal suppression; Laparoscopic surgery; IVF for infertility
  • Average diagnosis delay: 7-10 years from symptom onset

Clinical Pearls

"If She Has Cyclical Pain, Think Endometriosis": Any cyclical pelvic pain, especially with dysmenorrhoea, dyspareunia, or bowel/bladder symptoms around menses, should raise suspicion of endometriosis.

"Chocolate Cysts = Endometriomas": Ovarian endometriomas contain old blood ("chocolate" appearance). Ultrasound shows ground-glass echogenicity. Do NOT rupture during surgery (spill causes further endometriosis).

"Normal Scan Doesn't Exclude Endometriosis": Ultrasound may be normal, especially with superficial peritoneal disease. Laparoscopy is the gold standard for diagnosis.

"Excision > Ablation": Laparoscopic excision is preferred over ablation for better long-term outcomes and lower recurrence.

"Fertility Considerations Drive Management": In women wanting pregnancy, avoid prolonged hormonal suppression (doesn't treat infertility). Surgery or IVF may be needed.

Why This Matters Clinically

Endometriosis is common, debilitating, and underdiagnosed. Its impact on quality of life, mental health, and fertility is profound. Early recognition and appropriate referral can reduce the significant diagnostic delay and improve outcomes.[1,2]


2. Epidemiology

Incidence & Prevalence

ParameterData
Prevalence~10% of reproductive-age women
Infertile women25-50% have endometriosis
Chronic pelvic pain70% have endometriosis
AsymptomaticSome women are asymptomatic (incidental finding)

Risk Factors

FactorEffect
Early menarche↑ Risk
Short menstrual cycles↑ Risk
Family history7x increased risk in first-degree relatives
Nulliparity↑ Risk
Outflow obstruction↑ Risk (retrograde menstruation)
Increased oestrogen exposure↑ Risk

3. Pathophysiology

Theories of Origin

TheoryMechanism
Retrograde menstruationMost widely accepted; Menstrual tissue passes through fallopian tubes into pelvis
Coelomic metaplasiaPeritoneal cells transform into endometrial tissue
Lymphatic/Vascular spreadExplains distant sites (e.g., lungs, brain)
Stem cell theoryCirculating stem cells implant and differentiate

Pathological Features

FeatureDescription
Ectopic tissueEndometrial glands and stroma outside uterus
Hormone responsiveProliferates in response to oestrogen; Bleeds with menses
Inflammatory responseActivates macrophages; Produces cytokines; Causes adhesions
FibrosisRepeated bleeding → Fibrosis and scarring
Endometrioma formationOvarian cystic collections of old blood ("chocolate cyst")

4. Clinical Presentation

Symptoms

SymptomFrequencyNotes
Dysmenorrhoea80%Often severe; Progressive
Chronic pelvic pain70%Non-cyclical component
Dyspareunia40-50%Deep; Positional
Subfertility30-50%May be presenting complaint
Cyclical bowel symptomsVariableDyschezia; Rectal bleeding; Constipation
Cyclical urinary symptomsVariableDysuria; Haematuria
FatigueCommonChronic pain impact

Signs

SignNotes
Often normalEspecially with superficial disease
TendernessUterosacral ligaments; Pouch of Douglas
NodulesPalpable in rectovaginal septum or uterosacral ligaments
Fixed retroverted uterusAdhesions
Adnexal massEndometrioma

Red Flags

[!CAUTION]

  • Bowel obstruction symptoms
  • Ureteric involvement (hydronephrosis on imaging)
  • Rapidly enlarging ovarian mass (exclude malignancy)
  • Severe symptoms not responding to treatment

5. Clinical Examination

Pelvic Examination

ComponentFindings
SpeculumUsually normal; May see cervical lesions
BimanualTenderness; Nodules in uterosacral ligaments; Fixed uterus; Adnexal mass
RectovaginalNodules palpable in rectovaginal septum (deep infiltrating endometriosis)

General Examination

  • Usually normal
  • Assess for signs of anaemia (heavy/prolonged menses)

6. Investigations

Imaging

ModalityFindingsNotes
Transvaginal USSEndometriomas (ground-glass echogenicity); Kissing ovaries; Deep nodulesFirst-line; But may miss superficial disease
MRI PelvisDeep infiltrating endometriosis; Bladder/bowel involvementPre-operative planning for complex surgery

Blood Tests

TestNotes
CA-125May be elevated; NOT diagnostic (non-specific); May monitor treatment response
FBCCheck for anaemia

Definitive Diagnosis

InvestigationNotes
LaparoscopyGold standard for diagnosis; Visualise lesions; Take biopsies
HistologyEndometrial glands and stroma confirm diagnosis

rASRM Staging

StageDescription
I (Minimal)Isolated implants; No significant adhesions
II (Mild)Superficial implants <5 cm; No significant adhesions
III (Moderate)Deep implants; Small endometriomas; Mild adhesions
IV (Severe)Large endometriomas; Dense adhesions; Extensive disease

7. Management

Management Algorithm

             ENDOMETRIOSIS MANAGEMENT
                       ↓
┌───────────────────────────────────────────────────────────┐
│                SUSPECTED ENDOMETRIOSIS                    │
├───────────────────────────────────────────────────────────┤
│  Clinical suspicion (dysmenorrhoea, pelvic pain, etc.)   │
│  ➤ Transvaginal ultrasound                               │
│  ➤ Consider MRI if deep infiltrating disease suspected   │
│  ➤ Trial of empirical treatment if low suspicion of     │
│    alternative diagnosis                                  │
└───────────────────────────────────────────────────────────┘
                       ↓
┌───────────────────────────────────────────────────────────┐
│              FERTILITY NOT DESIRED                        │
├───────────────────────────────────────────────────────────┤
│  FIRST-LINE:                                              │
│  ➤ Analgesia: NSAIDs + Paracetamol                       │
│  ➤ Hormonal suppression:                                 │
│    • COCP (continuous tricycle regimen)                  │
│    • Progestogens (oral / Mirena IUS)                    │
│    • GnRH agonists (+ add-back HRT to prevent bone loss) │
│                                                           │
│  SECOND-LINE / REFRACTORY:                                │
│  ➤ Laparoscopic surgery (excision preferred over ablation)│
│  ➤ Hysterectomy ± BSO (definitive; selected cases)       │
└───────────────────────────────────────────────────────────┘
                       ↓
┌───────────────────────────────────────────────────────────┐
│               FERTILITY DESIRED                           │
├───────────────────────────────────────────────────────────┤
│  ➤ Hormonal suppression does NOT treat infertility       │
│                                                           │
│  MILD/MODERATE DISEASE:                                   │
│  ➤ Laparoscopic excision may improve fertility           │
│  ➤ Consider expectant management (natural conception)   │
│                                                           │
│  SEVERE DISEASE / FAILED CONCEPTION:                      │
│  ➤ IVF/ICSI                                              │
│  ➤ Endometrioma &gt;4 cm: Consider excision before IVF     │
│                                                           │
│  ⚠️ Avoid prolonged hormonal suppression if trying to   │
│    conceive — doesn't improve fertility                  │
└───────────────────────────────────────────────────────────┘
                       ↓
┌───────────────────────────────────────────────────────────┐
│            DEEP INFILTRATING ENDOMETRIOSIS                │
├───────────────────────────────────────────────────────────┤
│  ➤ Multidisciplinary approach                            │
│  ➤ MRI for surgical planning                             │
│  ➤ Specialist centre referral                            │
│  ➤ Bowel/bladder resection may be required               │
│  ➤ Ureteric stenting if obstruction                      │
└───────────────────────────────────────────────────────────┘

Hormonal Options

TreatmentMechanismNotes
COCP (continuous)Suppresses ovulation; Thins endometriumFirst-line; Flexible regimens
ProgestogensDecidualisation; AtrophyOral (norethisterone); POP; Mirena IUS
GnRH agonistsInduced menopauseGoserelin, Leuprorelin; Add-back HRT needed if >6 months
GnRH antagonistsSimilar to agonistsElagolix (newer)

8. Complications
ComplicationNotes
InfertilityDue to adhesions, distorted anatomy, inflammation
Ovarian cyst rupture/torsionEndometrioma complications
Bowel obstructionDeep infiltrating disease
Ureteric obstructionHydronephrosis
Chronic painSignificant quality of life impact
Perioperative morbidityComplex surgery for deep disease

9. Prognosis & Outcomes
FactorOutcome
Recurrence after surgery20-40% at 5 years
Hormonal treatmentSuppresses symptoms; Doesn't cure or improve fertility
IVF successLive birth rates similar to other causes of infertility
MenopauseSymptoms usually improve

10. Evidence & Guidelines

Key Guidelines

GuidelineOrganisationYearKey Points
Endometriosis: Diagnosis and Management (NG73)NICE2017Diagnosis, medical and surgical management
ESHRE GuidelineESHRE2022European guideline; Fertility focus

11. Patient/Layperson Explanation

What is endometriosis?

Endometriosis is a condition where tissue similar to the lining of the womb grows in other places, like the ovaries, bowel, or pelvis. This tissue responds to hormones and bleeds during your period, but the blood has nowhere to go, causing pain and scarring.

What are the symptoms?

  • Painful periods
  • Pelvic pain (especially around your period)
  • Pain during or after sex
  • Difficulty getting pregnant
  • Bowel or bladder symptoms during your period

How is it diagnosed?

An ultrasound can sometimes show cysts, but often the only way to diagnose endometriosis for certain is with a keyhole operation (laparoscopy) where the doctor looks inside your pelvis.

How is it treated?

  • Painkillers (like ibuprofen)
  • Hormonal treatments (pill, coil, injections) to stop periods and slow the disease
  • Surgery to remove the endometriosis tissue
  • IVF if you're trying for a baby and other treatments haven't worked

Can it be cured?

Endometriosis is a long-term condition. Treatments can help control symptoms, but it often comes back. Symptoms usually improve after menopause.


12. References
  1. NICE. Endometriosis: diagnosis and management (NG73). 2017. nice.org.uk/guidance/ng73

13. Examination Focus

High-Yield Exam Topics

TopicKey Points
Prevalence10% of reproductive women
SymptomsDysmenorrhoea, pelvic pain, dyspareunia, subfertility
Gold standard diagnosisLaparoscopy + Histology
Treatment (no fertility wish)Hormonal suppression; Surgery
Treatment (fertility wish)Avoid prolonged hormonal suppression; Surgery; IVF
EndometriomaChocolate cyst; Ground-glass on USS

Sample Viva Question

Q: A 28-year-old woman has severe dysmenorrhoea and dyspareunia. How would you investigate and manage suspected endometriosis?

Model Answer: I would take a detailed history including cyclical nature of symptoms, bowel/bladder symptoms, and fertility wishes. Examination: bimanual and rectovaginal for nodules and tenderness. Initial investigation: Transvaginal ultrasound to look for endometriomas (ground glass appearance) or deep infiltrating disease. MRI if complex disease suspected a

definitive diagnosis requires laparoscopy with biopsy. Management depends on fertility wishes:

  • Not seeking pregnancy: Trial of COCP continuous regimen or progestogens (Mirena IUS); NSAIDs for pain. If refractory, laparoscopic excision.
  • Seeking pregnancy: Hormonal suppression doesn't improve fertility. Consider laparoscopic excision for mild-moderate disease. IVF for severe disease or failed conception.

Last Reviewed: 2025-12-24 | MedVellum Editorial Team

Last updated: 2025-12-24

At a Glance

EvidenceHigh
Last Updated2025-12-24

Red Flags

  • Bowel obstruction
  • Ureteric involvement / Hydronephrosis
  • Severe pain not responding to treatment
  • Ovarian mass (exclude malignancy)

Clinical Pearls

  • **"Normal Scan Doesn't Exclude Endometriosis"**: Ultrasound may be normal, especially with superficial peritoneal disease. Laparoscopy is the gold standard for diagnosis.
  • Ablation"**: Laparoscopic excision is preferred over ablation for better long-term outcomes and lower recurrence.
  • **"Fertility Considerations Drive Management"**: In women wanting pregnancy, avoid prolonged hormonal suppression (doesn't treat infertility). Surgery or IVF may be needed.
  • - Bowel obstruction symptoms
  • - Ureteric involvement (hydronephrosis on imaging)

Guidelines

  • NICE Guidelines
  • BTS Guidelines
  • RCUK Guidelines