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Orthopaedics
Paediatrics

Clubfoot (Talipes Equinovarus)

High EvidenceUpdated: 2025-12-22

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Red Flags

  • Syndromic clubfoot (spina bifida, arthrogryposis, Down syndrome)
  • Rigid deformity not improving with casting
  • Neuromuscular cause suspected
  • Relapse after initial correction
  • Skin complications from casting
Overview

Clubfoot (Talipes Equinovarus)

1. Topic Overview

Summary

Clubfoot (congenital talipes equinovarus, CTEV) is a congenital foot deformity affecting 1 in 1,000 live births, making it one of the most common musculoskeletal birth defects. The deformity involves four components remembered by the mnemonic CAVE: Cavus, Adductus, Varus, and Equinus. The Ponseti method of serial casting and percutaneous Achilles tenotomy has revolutionised treatment, with success rates exceeding 95%. Boots-and-bar bracing for 4 years after correction is essential to prevent relapse.

Key Facts

  • Definition: Congenital foot deformity with forefoot adduction, hindfoot varus and equinus
  • Incidence: 1 per 1,000 live births (varies by ethnicity)
  • Sex Ratio: Male:Female 2:1
  • Laterality: Bilateral in 50% of cases
  • Treatment:Ponseti method is gold standard (95%+ success rate)
  • Key to Success: Brace compliance (boots-and-bar for 4 years)

Clinical Pearls

"CAVE" Mnemonic: The four deformity components in order of correction are: Cavus (high arch), Adductus (forefoot adducted), Varus (hindfoot inverted), Equinus (ankle plantarflexed). Correct CAVE in order — C-A-V first with casting, E last with tenotomy.

Tenotomy in 90%: Approximately 90% of clubfeet require percutaneous Achilles tenotomy after casting to correct the equinus. This is done in clinic under local anaesthesia and is definitive.

Relapse is Parent-Dependent: Relapse occurs in 40% if bracing is not used correctly. The single biggest predictor of relapse is brace non-compliance. Educate parents extensively.

Why This Matters Clinically

Clubfoot, if untreated, leads to permanent disability, painful walking on the outer edge of the foot, and difficulties with footwear. The Ponseti method has transformed outcomes — with appropriate treatment, children achieve normal functional feet. Early referral to a Ponseti-trained specialist is essential.


2. Epidemiology

Incidence & Prevalence

  • Incidence: 1 per 1,000 live births (worldwide average)
  • Ethnic variation: Higher in Polynesian populations (6-7/1000), lower in East Asian populations (0.3-0.5/1000)
  • UK incidence: Approximately 1,000 babies per year
  • Trend: Stable incidence

Demographics

FactorDetails
SexMale:Female 2:1
SideRight = Left = Bilateral (each ~33%)
Bilateral50% of cases
Family History25% have affected family member

Risk Factors

Non-Modifiable:

  • Male sex (2x risk)
  • Family history (25% positive)
  • Genetic syndromes (arthrogryposis, spina bifida)
  • Oligohydramnios (theoretical — compression)

Modifiable:

Risk FactorNotes
Maternal smokingSome evidence of increased risk
Amniocentesis <15 weeksAssociated with clubfoot

3. Pathophysiology

Mechanism

Exact Aetiology Unknown — Multifactorial:

Theories:

  1. Germ plasm defect: Primary developmental abnormality of talus
  2. Neurogenic: Abnormal muscle innervation
  3. Vascular: Abnormal anterior tibial artery development
  4. Arrested development: Foot fails to derotate in utero
  5. Genetic: Polygenic inheritance pattern

Pathological Anatomy:

  • Talus: Short neck, medially deviated
  • Navicular: Displaced medially onto medial malleolus
  • Calcaneus: Inverted, adducted, rotated medially
  • Cuboid: Displaced medially
  • Medial structures: Contracted (tibialis posterior, Achilles, ligaments)

Classification (CAVE Mnemonic)

ComponentDeformityAnatomical Basis
CavusHigh longitudinal archFirst metatarsal plantarflexed
AdductusForefoot pointed inwardTight medial structures
VarusHeel turned inwardCalcaneus inverted under talus
EquinusFoot plantarflexedShort Achilles tendon

Classification by Aetiology

TypeFeaturesPrognosis
IdiopathicIsolated clubfoot, otherwise normal childExcellent with Ponseti
NeurogenicSpina bifida, myelomeningoceleVariable; higher relapse
SyndromicArthrogryposis, chromosomal anomaliesOften resistant to treatment
PositionalVery flexible, correctableExcellent; minimal treatment

4. Clinical Presentation

Symptoms

Prenatal:

At Birth:

Untreated (Historical):

Signs

Red Flags

[!CAUTION] Red Flags — Suggest complex or syndromic clubfoot:

  • Associated spinal abnormality (sacral dimple, tuft of hair — spina bifida)
  • Hip abnormality (DDH — occurs in 3-5% with clubfoot)
  • Multiple joint contractures (arthrogryposis)
  • Severe rigidity not responding to initial casting
  • Neurological abnormality (check spine MRI)

Often detected on antenatal ultrasound (18-20 week scan)
Common presentation.
Parents may be counselled antenatally
Common presentation.
5. Clinical Examination

Structured Approach

General:

  • Full newborn examination (exclude syndromic features)
  • Spine examination (sacral dimple, tuft of hair)
  • Hip examination (Barlow/Ortolani for DDH)
  • Both feet and legs examined

Specific Clubfoot Examination:

  • Assess each CAVE component
  • Test passibility of deformity (flexible vs rigid)
  • Compare to contralateral side (if unilateral)
  • Assess calf size (typically smaller on affected side)

Special Tests

TestTechniquePositive FindingSignificance
Pirani Score6-point scoring system0 = fully corrected; 6 = severeMonitor progress of treatment
Dimeglio ScoreAlternative severity scoreGrade I-IVPredict treatment difficulty
Passive CorrectionAttempt to correct each componentRigid = concerningSyndromic clubfoot if very rigid
Hip ExaminationBarlow and OrtolaniInstabilityDDH association
Spine ExaminationInspect for dimple/tuftPresentPossible spina bifida

Pirani Scoring System

Hindfoot (3 points):

  • Posterior crease severity (0-1)
  • Emptiness of heel (0-1)
  • Rigidity of equinus (0-1)

Midfoot (3 points):

  • Medial crease severity (0-1)
  • Lateral border curvature (0-1)
  • Head of talus coverage (0-1)

Total: 0 (normal) to 6 (severe)


6. Investigations

First-Line (Bedside)

  • Clinical examination is diagnostic
  • Pirani or Dimeglio scoring for severity and monitoring
  • Hip examination for associated DDH

Laboratory Tests

TestExpected FindingPurpose
Usually not required—Diagnosis is clinical
KaryotypeIf syndromic featuresGenetic diagnosis

Imaging

ModalityFindingsIndication
X-rayRarely needed for diagnosisMay assess talocalcaneal angle if needed
Ultrasound HipDDH screeningAll babies with clubfoot
MRI SpineSpinal dysraphismIf sacral dimple, neurological signs
Prenatal UltrasoundClubfoot visible at 18-20 weeksAntenatal detection

Diagnostic Criteria

Clinical diagnosis based on:

  1. Presence of CAVE deformity at birth
  2. Rigidity of deformity (cannot passively correct)
  3. Exclusion of positional talipes (which is flexible)

7. Management

Management Algorithm

CLUBFOOT MANAGEMENT (PONSETI METHOD)
              ↓
┌─────────────────────────────────────────────────────┐
│              PHASE 1: CASTING                       │
│                                                     │
│ • Weekly manipulation and above-knee casts          │
│ • Correct CAVE in order: Cavus → Adductus → Varus   │
│ • Do NOT correct equinus during casting             │
│ • Typically 4-6 casts over 4-6 weeks               │
│ • Monitor with Pirani score                         │
└─────────────────────────────────────────────────────┘
              ↓
┌─────────────────────────────────────────────────────┐
│        PHASE 2: ACHILLES TENOTOMY                   │
│                                                     │
│ • Percutaneous tenotomy (90% of patients)           │
│ • Done when CAV corrected but E persists            │
│ • In clinic, local anaesthesia, small blade         │
│ • Final cast for 3 weeks post-tenotomy              │
│ • Tendon regenerates in lengthened position         │
└─────────────────────────────────────────────────────┘
              ↓
┌─────────────────────────────────────────────────────┐
│        PHASE 3: BRACING (CRITICAL!)                 │
│                                                     │
│ Boots-and-Bar (Foot Abduction Brace - FAB):         │
│ • Full-time (23 hours/day) for 3 months             │
│ • Then nights + naps until age 4 years              │
│ • Set at 70° external rotation (affected side)      │
│                                                     │
│ ⚠ COMPLIANCE IS KEY — 40% relapse if non-compliant │
└─────────────────────────────────────────────────────┘
              ↓
         RELAPSE?
              ↓
┌─────────────────────────────────────────────────────┐
│ Mild: Repeat casting series                         │
│ Equinus: Repeat Achilles tenotomy                   │
│ Dynamic supination (walking age): Tibialis anterior │
│          transfer (after age 2.5)                   │
│ Severe/Complex: Posteromedial release (rare now)    │
└─────────────────────────────────────────────────────┘

Conservative Management (Ponseti Method)

Casting Phase:

  • Begin as soon as possible (ideally within 1-2 weeks of birth)
  • Weekly above-knee plaster casts
  • Manipulation follows specific Ponseti technique
  • Correct cavus first (supinate forefoot to align with hindfoot)
  • Then correct adductus and varus by abducting foot with counter-pressure on lateral talar head (NEVER pronate)
  • Do NOT attempt to correct equinus during casting — must be last

Achilles Tenotomy:

  • Indicated when CAV corrected but foot cannot dorsiflex beyond neutral
  • Done percutaneously in clinic or minor procedure room
  • Local anaesthesia (EMLA cream + subcutaneous LA)
  • Complete division of Achilles tendon
  • Final above-knee cast for 3 weeks
  • Tendon regenerates in lengthened position

Bracing Phase:

  • Boots attached to bar at 70° external rotation (affected side), 40° (normal side)
  • Bar width = shoulder width
  • Full-time for first 3 months
  • Then nights and naps until age 4 years
  • Total duration: ~4 years

Surgical Management

Indications (Now Rare <5%):

  • Failure of Ponseti method
  • Severe rigid syndromic clubfoot
  • Late-presenting neglected clubfoot
  • Recurrent relapse despite proper bracing

Procedures:

ProcedureDescriptionIndication
Tibialis Anterior TransferTransfer TAT to lateral cuneiformDynamic supination >2.5 years
Posteromedial ReleaseExtensive soft tissue releaseHistorically used; rarely now
External FixationFrame correctionNeglected/recurrent deformity
OsteotomiesBony correctionOlder child with rigid deformity

8. Complications

Treatment-Related

ComplicationCausePrevention/Management
Cast soresPressure from castProper casting technique, check after 24 hours
Cast slippingInadequate mouldingAbove-knee cast essential
Rocker-bottomForced dorsiflexion too earlyNever try to correct equinus before CAV
Bleeding post-tenotomyTenotomy complicationPressure, elevation; rarely significant

Relapse (Most Common Long-Term Issue)

  • Incidence: 40% if non-compliant with bracing; <10% if compliant
  • Risk factors: Non-compliance, syndromic clubfoot, severe initial deformity
  • Management: Re-casting, repeat tenotomy, TAT transfer as needed

Late Complications

  • Persistent calf atrophy: Common; usually not functionally significant
  • Foot size difference: Affected foot usually 0.5-1 size smaller
  • Residual deformity: May require late osteotomies
  • Osteoarthritis: Rare if well-treated

9. Prognosis & Outcomes

Natural History

Untreated clubfoot leads to walking on the lateral border of the foot, painful calluses, difficulty with footwear, and significant disability. With proper Ponseti treatment, outcomes are excellent.

Outcomes with Treatment

VariableOutcome
Ponseti success rate>95% functional correction
Tenotomy rate90% require tenotomy
Relapse with good compliance5-10%
Relapse with poor compliance40%
Long-term functionNear-normal gait and activity

Prognostic Factors

Good Prognosis:

  • Idiopathic clubfoot
  • Early treatment
  • Good brace compliance
  • Supple deformity

Poor Prognosis:

  • Syndromic or neurogenic clubfoot
  • Delayed treatment
  • Poor brace compliance
  • Severe rigid deformity (high Pirani score)

10. Evidence & Guidelines

Key Guidelines

  1. Ponseti International Association — Provides training and standards for Ponseti method worldwide.

  2. POSNA (Pediatric Orthopaedic Society of North America) — Endorses Ponseti method as gold standard.

Landmark Studies

Ponseti (1963-1996) — Original technique description

  • Iowa long-term outcomes
  • Key finding: Non-surgical correction possible in majority
  • Clinical Impact: Revolutionised clubfoot treatment worldwide

Morcuende et al. (2004) — Compliance study

  • Key finding: Relapse directly related to brace non-compliance
  • Clinical Impact: Emphasised critical importance of parent education

Dobbs et al. (2004) — Long-term outcomes

  • 30-year follow-up of Ponseti patients
  • Key finding: Excellent functional outcomes maintained
  • Clinical Impact: Confirmed durability of Ponseti correction

Evidence Strength

InterventionLevelKey Evidence
Ponseti method1aMultiple cohort studies, meta-analyses
Achilles tenotomy2aLarge case series
Bracing (FAB)2aComparative studies showing relapse prevention
TAT for relapse2bCase series

11. Patient/Layperson Explanation

What is Clubfoot?

Clubfoot is a condition where a baby is born with one or both feet turned inward and downward. The medical name is "talipes equinovarus" or "CTEV." It's one of the most common birth defects affecting bones and muscles, occurring in about 1 in every 1,000 babies.

Why does it matter?

Without treatment, clubfoot would make it very difficult to walk normally. Children would have to walk on the outside of their feet, causing pain and disability. The good news is that with proper treatment, children with clubfoot can have completely normal feet that work just like everyone else's.

How is it treated?

Treatment uses a method called the Ponseti method, which is very effective:

  1. Casting (4-6 weeks): The foot is gently stretched into a better position, and a plaster cast is applied. This is done weekly, with each cast getting closer to the correct position.

  2. Achilles tenotomy (9 out of 10 babies): A small cut is made to lengthen the tight tendon at the back of the ankle. This is done in clinic and isn't as scary as it sounds. A final cast is worn for 3 weeks after.

  3. Boots-and-bar bracing (4 years): This is the most important part. Special boots attached to a bar are worn to keep the feet in the corrected position:

    • Full-time for the first 3 months
    • Then during sleep and naps until age 4

What to expect

  • Treatment starts soon after birth
  • Weekly clinic visits for 4-6 weeks
  • Your child will have a small scar on the back of the ankle
  • The affected foot and calf will always be slightly smaller — this is normal
  • With proper brace wearing, your child should have a completely normal, functional foot

When to seek help

Contact your clinic if:

  • The cast becomes loose or slips
  • You see any skin marks or sores
  • Your child seems in pain with the cast
  • You're having trouble with the braces
  • The foot seems to be turning back inward (relapse)

12. References

Primary Guidelines

  1. Ponseti International Association. Clubfoot: Ponseti Management. 3rd Edition. 2021.

Key Trials

  1. Ponseti IV, Smoley EN. The classic: congenital club foot: the results of treatment. Clin Orthop Relat Res. 2009;467(5):1133-1145. PMID: 19219518

  2. Morcuende JA, Dolan LA, Dietz FR, Ponseti IV. Radical reduction in the rate of extensive corrective surgery for clubfoot using the Ponseti method. Pediatrics. 2004;113(2):376-380. PMID: 14754952

  3. Dobbs MB, Rudzki JR, Purcell DB, et al. Factors predictive of outcome after use of the Ponseti method for the treatment of idiopathic clubfeet. J Bone Joint Surg Am. 2004;86(1):22-27. PMID: 14711941

Further Resources

  • Ponseti International Association: ponseti.info
  • Steps Charity Worldwide (UK): steps-charity.org.uk


Medical Disclaimer: MedVellum content is for educational purposes and clinical reference. Clinical decisions should account for individual patient circumstances. Always consult appropriate guidelines and specialists for patient care.

Last updated: 2025-12-22

At a Glance

EvidenceHigh
Last Updated2025-12-22

Red Flags

  • Syndromic clubfoot (spina bifida, arthrogryposis, Down syndrome)
  • Rigid deformity not improving with casting
  • Neuromuscular cause suspected
  • Relapse after initial correction
  • Skin complications from casting

Clinical Pearls

  • **Tenotomy in 90%**: Approximately 90% of clubfeet require percutaneous Achilles tenotomy after casting to correct the equinus. This is done in clinic under local anaesthesia and is definitive.
  • **Relapse is Parent-Dependent**: Relapse occurs in 40% if bracing is not used correctly. The single biggest predictor of relapse is brace non-compliance. Educate parents extensively.
  • **Red Flags** — Suggest complex or syndromic clubfoot:
  • - Associated spinal abnormality (sacral dimple, tuft of hair — spina bifida)
  • - Hip abnormality (DDH — occurs in 3-5% with clubfoot)

Guidelines

  • NICE Guidelines
  • BTS Guidelines
  • RCUK Guidelines